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1.
Kidney dysfunction induced by a sucrose-rich diet in rat involves mitochondria ROS generation, cardiolipin changes, and the decline of autophagy protein markers.
Am J Physiol Renal Physiol
; 318(1): F53-F66, 2020 01 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-31657248
2.
Cytochrome c release from rat liver mitochondria is compromised by increased saturated cardiolipin species induced by sucrose feeding.
Am J Physiol Endocrinol Metab
; 309(9): E777-86, 2015 Nov 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-26353385
3.
Lipotoxicity, glucotoxicity and some strategies to protect vascular smooth muscle cell against proliferative phenotype in metabolic syndrome.
Food Chem Toxicol
; 172: 113546, 2023 Feb.
Artículo
en Inglés
| MEDLINE | ID: mdl-36513245
4.
Endogenous Liver Protections Against Lipotoxicity and Oxidative Stress to Avoid the Progression of Non-alcoholic Fatty Liver to more Serious Disease.
Curr Mol Med
; 22(5): 401-420, 2022.
Artículo
en Inglés
| MEDLINE | ID: mdl-34931979
5.
Palmitoyl-CoA effect on cytochrome c release, a key process of apoptosis, from liver mitochondria of rat with sucrose diet-induced obesity.
Food Chem Toxicol
; 154: 112351, 2021 Aug.
Artículo
en Inglés
| MEDLINE | ID: mdl-34171418
6.
Reactive Oxygen Species from NADPH Oxidase and Mitochondria Participate in the Proliferation of Aortic Smooth Muscle Cells from a Model of Metabolic Syndrome.
Oxid Med Cell Longev
; 2018: 5835072, 2018.
Artículo
en Inglés
| MEDLINE | ID: mdl-30671170
7.
Cell Death and Heart Failure in Obesity: Role of Uncoupling Proteins.
Oxid Med Cell Longev
; 2016: 9340654, 2016.
Artículo
en Inglés
| MEDLINE | ID: mdl-27642497
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