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1.
Dynamics of plasma thymidine kinase activity in metastatic melanoma reflects immune checkpoint inhibitor efficacy.
Acta Oncol
; 61(9): 1116-1120, 2022 09.
Artículo
en Inglés
| MEDLINE | ID: mdl-36083107
2.
Treatment Patterns and Efficacy of Chemotherapy After Pembrolizumab in Advanced Urothelial Cancer-a Real-World Study in the pre-Antibody-Drug Conjugate Era.
Clin Genitourin Cancer
; 21(6): e438-e448, 2023 12.
Artículo
en Inglés
| MEDLINE | ID: mdl-37308329
3.
Phase II LEAP-004 Study of Lenvatinib Plus Pembrolizumab for Melanoma With Confirmed Progression on a Programmed Cell Death Protein-1 or Programmed Death Ligand 1 Inhibitor Given as Monotherapy or in Combination.
J Clin Oncol
; 41(1): 75-85, 2023 01 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-35867951
4.
Proliferation and Immune Response Gene Signatures Associated with Clinical Outcome to Immunotherapy and Targeted Therapy in Metastatic Cutaneous Malignant Melanoma.
Cancers (Basel)
; 14(15)2022 Jul 22.
Artículo
en Inglés
| MEDLINE | ID: mdl-35892846
5.
Plasma Thymidine Kinase Activity as a Novel Biomarker in Metastatic Melanoma Patients Treated with Immune Checkpoint Inhibitors.
Cancers (Basel)
; 14(3)2022 Jan 29.
Artículo
en Inglés
| MEDLINE | ID: mdl-35158970
6.
[Overview of immune-related side effects from immune checkpoint inhibitors. Part 1: Gastrointestinal, lung and kidney toxicity]. / Immunrelaterade biverkningar i tarm, lever, lungor och njurar.
Lakartidningen
; 1182021 11 18.
Artículo
en Sueco
| MEDLINE | ID: mdl-35080767
7.
Inflammation and Apolipoproteins Are Potential Biomarkers for Stratification of Cutaneous Melanoma Patients for Immunotherapy and Targeted Therapy.
Cancer Res
; 81(9): 2545-2555, 2021 05 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-33574091
8.
Combining ERBB family and MET inhibitors is an effective therapeutic strategy in cutaneous malignant melanoma independent of BRAF/NRAS mutation status.
Cell Death Dis
; 10(9): 663, 2019 09 10.
Artículo
en Inglés
| MEDLINE | ID: mdl-31506424
9.
Silencing FLI or targeting CD13/ANPEP lead to dephosphorylation of EPHA2, a mediator of BRAF inhibitor resistance, and induce growth arrest or apoptosis in melanoma cells.
Cell Death Dis
; 8(8): e3029, 2017 08 31.
Artículo
en Inglés
| MEDLINE | ID: mdl-29048432
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