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1.
c-Jun signaling during initial HSV-1 infection modulates latency to enhance later reactivation in addition to directly promoting the progression to full reactivation.
J Virol
; 98(2): e0176423, 2024 Feb 20.
Artículo
en Inglés
| MEDLINE | ID: mdl-38193709
2.
The Intersection of Innate Immune Pathways with the Latent Herpes Simplex Virus Genome.
J Virol
; 97(5): e0135222, 2023 05 31.
Artículo
en Inglés
| MEDLINE | ID: mdl-37129520
3.
PML-NB-dependent type I interferon memory results in a restricted form of HSV latency.
EMBO Rep
; 22(9): e52547, 2021 09 06.
Artículo
en Inglés
| MEDLINE | ID: mdl-34197022
4.
c-Jun Signaling During Initial HSV-1 Infection Modulates Latency to Enhance Later Reactivation in addition to Directly Promoting the Progression to Full Reactivation.
bioRxiv
; 2023 Nov 10.
Artículo
en Inglés
| MEDLINE | ID: mdl-37986840
5.
The secret life of degradative lysosomes in axons: delivery from the soma, enzymatic activity, and local autophagic clearance.
Autophagy
; 16(1): 167-168, 2020 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-31533518
6.
Neuronal hyperexcitability is a DLK-dependent trigger of herpes simplex virus reactivation that can be induced by IL-1.
Elife
; 92020 12 22.
Artículo
en Inglés
| MEDLINE | ID: mdl-33350386
7.
Neuronal Soma-Derived Degradative Lysosomes Are Continuously Delivered to Distal Axons to Maintain Local Degradation Capacity.
Cell Rep
; 28(1): 51-64.e4, 2019 07 02.
Artículo
en Inglés
| MEDLINE | ID: mdl-31269450
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