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1.
IGF1R constitutive activation expands luminal progenitors and influences lineage differentiation during breast tumorigenesis.
Dev Biol
; 463(1): 77-87, 2020 07 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-32376245
2.
Come on baby, let's do the twist: the kinematics of killing in loggerhead shrikes.
Biol Lett
; 14(9)2018 09.
Artículo
en Inglés
| MEDLINE | ID: mdl-30185607
3.
Lack of interaction between ErbB2 and insulin receptor substrate signaling in breast cancer.
Cell Commun Signal
; 14(1): 25, 2016 10 21.
Artículo
en Inglés
| MEDLINE | ID: mdl-27765041
4.
Expression of Six1 in luminal breast cancers predicts poor prognosis and promotes increases in tumor initiating cells by activation of extracellular signal-regulated kinase and transforming growth factor-beta signaling pathways.
Breast Cancer Res
; 14(4): R100, 2012 Jul 05.
Artículo
en Inglés
| MEDLINE | ID: mdl-22765220
5.
Epithelial-mesenchymal transition in cancer: parallels between normal development and tumor progression.
J Mammary Gland Biol Neoplasia
; 15(2): 117-34, 2010 Jun.
Artículo
en Inglés
| MEDLINE | ID: mdl-20490631
6.
Role of IGF1R in Breast Cancer Subtypes, Stemness, and Lineage Differentiation.
Front Endocrinol (Lausanne)
; 6: 59, 2015.
Artículo
en Inglés
| MEDLINE | ID: mdl-25964777
7.
Contribution of xanthine oxidoreductase to mammary epithelial and breast cancer cell differentiation in part modulates inhibitor of differentiation-1.
Mol Cancer Res
; 9(9): 1242-54, 2011 Sep.
Artículo
en Inglés
| MEDLINE | ID: mdl-21775420
8.
Homeoprotein Six1 increases TGF-beta type I receptor and converts TGF-beta signaling from suppressive to supportive for tumor growth.
Cancer Res
; 70(24): 10371-80, 2010 Dec 15.
Artículo
en Inglés
| MEDLINE | ID: mdl-21056993
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