Búsqueda | Biblioteca Virtual en Salud

1.

FE65 and FE65L1 amyloid precursor protein-binding protein compound null mice display adult-onset cataract and muscle weakness.

Suh, Jaehong; Moncaster, Juliet A; Wang, Lirong; Hafeez, Imran; Herz, Joachim; Tanzi, Rudolph E; Goldstein, Lee E; Guénette, Suzanne Y.

FASEB J ; 29(6): 2628-39, 2015 Jun.

Artículo en Inglés | MEDLINE | ID: mdl-25757569

2.

Amyloid beta a4 precursor protein-binding family B member 1 (FE65) interactomics revealed synaptic vesicle glycoprotein 2A (SV2A) and sarcoplasmic/endoplasmic reticulum calcium ATPase 2 (SERCA2) as new binding proteins in the human brain.

Nensa, Fabian M; Neumann, Martin H D; Schrötter, Andreas; Przyborski, Andre; Mastalski, Thomas; Susdalzew, Sergej; Looße, Christina; Helling, Stefan; El Magraoui, Fouzi; Erdmann, Ralf; Meyer, Helmut E; Uszkoreit, Julian; Eisenacher, Martin; Suh, Jaehong; Guénette, Suzanne Y; Röhner, Nelli; Kögel, Donat; Theiss, Carsten; Marcus, Katrin; Müller, Thorsten.

Mol Cell Proteomics ; 13(2): 475-88, 2014 Feb.

Artículo en Inglés | MEDLINE | ID: mdl-24284412

3.

A role for FE65 in controlling GnRH-1 neurogenesis.

Forni, Paolo E; Fornaro, Michele; Guénette, Suzanne; Wray, Susan.

J Neurosci ; 31(2): 480-91, 2011 Jan 12.

Artículo en Inglés | MEDLINE | ID: mdl-21228158

4.

FE65 proteins regulate NMDA receptor activation-induced amyloid precursor protein processing.

Suh, Jaehong; Lyckman, Alvin; Wang, Lirong; Eckman, Elizabeth A; Guénette, Suzanne Y.

J Neurochem ; 119(2): 377-88, 2011 Oct.

Artículo en Inglés | MEDLINE | ID: mdl-21824144

5.

Hypoxic ischemia and proteasome dysfunction alter tau isoform ratio by inhibiting exon 10 splicing.

Suh, Jaehong; Im, Doo Soon; Moon, Gyeong Joon; Ryu, Keun Sil; de Silva, Rohan; Choi, In Sun; Lees, Andrew J; Guénette, Suzanne Y; Tanzi, Rudolph E; Gwag, Byoung Joo.

J Neurochem ; 114(1): 160-70, 2010 Jul.

Artículo en Inglés | MEDLINE | ID: mdl-20374429

6.

APP Protein Family Signaling at the Synapse: Insights from Intracellular APP-Binding Proteins.

Guénette, Suzanne; Strecker, Paul; Kins, Stefan.

Front Mol Neurosci ; 10: 87, 2017.

Artículo en Inglés | MEDLINE | ID: mdl-28424586

7.

FE65 and FE65L1 share common synaptic functions and genetically interact with the APP family in neuromuscular junction formation.

Strecker, Paul; Ludewig, Susann; Rust, Marco; Mundinger, Tabea A; Görlich, Andreas; Krächan, Elisa G; Mehrfeld, Christina; Herz, Joachim; Korte, Martin; Guénette, Suzanne Y; Kins, Stefan.

Sci Rep ; 6: 25652, 2016 05 11.

Artículo en Inglés | MEDLINE | ID: mdl-27734846

8.

A role for APP in motility and transcription?

Guénette, Suzanne Y.

Trends Pharmacol Sci ; 23(5): 203-5; discussion 205-6, 2002 May.

Artículo en Inglés | MEDLINE | ID: mdl-12007991

9.

Mechanisms of Abeta clearance and catabolism.

Guénette, Suzanne Y.

Neuromolecular Med ; 4(3): 147-60, 2003.

Artículo en Inglés | MEDLINE | ID: mdl-14716023

10.

No major role for insulin-degrading enzyme in antigen presentation by MHC molecules.

Culina, Slobodan; Mauvais, François-Xavier; Hsu, Hsiang-Ting; Burgevin, Anne; Guénette, Suzanne; Moser, Anna; van Endert, Peter.

PLoS One ; 9(2): e88365, 2014.

Artículo en Inglés | MEDLINE | ID: mdl-24516642

11.

FE65 as a link between VLDLR and APP to regulate their trafficking and processing.

Dumanis, Sonya B; Chamberlain, Kelly A; Jin Sohn, Yoo; Jin Lee, Young; Guénette, Suzanne Y; Suzuki, Toshiharu; Mathews, Paul M; Pak, Daniel Ts; Rebeck, G William; Suh, Yoo-Hun; Park, Hee-Sae; Hoe, Hyang-Sook.

Mol Neurodegener ; 7: 9, 2012 Mar 19.

Artículo en Inglés | MEDLINE | ID: mdl-22429478

12.

Astrocytes: a cellular player in Abeta clearance and degradation.

Guénette, Suzanne Y.

Trends Mol Med ; 9(7): 279-80, 2003 Jul.

Artículo en Inglés | MEDLINE | ID: mdl-12900212

13.

HtrA2 regulates beta-amyloid precursor protein (APP) metabolism through endoplasmic reticulum-associated degradation.

Huttunen, Henri J; Guénette, Suzanne Y; Peach, Camilla; Greco, Christopher; Xia, Weiming; Kim, Doo Yeon; Barren, Cory; Tanzi, Rudolph E; Kovacs, Dora M.

J Biol Chem ; 282(38): 28285-95, 2007 Sep 21.

Artículo en Inglés | MEDLINE | ID: mdl-17684015

14.

FE65 interaction with the ApoE receptor ApoEr2.

Hoe, Hyang-Sook; Magill, Laura Ann; Guenette, Suzanne; Fu, Zhanyan; Vicini, Stefano; Rebeck, G William.

J Biol Chem ; 281(34): 24521-30, 2006 Aug 25.

Artículo en Inglés | MEDLINE | ID: mdl-16638748

15.

Essential roles for the FE65 amyloid precursor protein-interacting proteins in brain development.

Guénette, Suzanne; Chang, Yang; Hiesberger, Thomas; Richardson, James A; Eckman, Christopher B; Eckman, Elizabeth A; Hammer, Robert E; Herz, Joachim.

EMBO J ; 25(2): 420-31, 2006 Jan 25.

Artículo en Inglés | MEDLINE | ID: mdl-16407979

16.

Low-density lipoprotein receptor-related protein levels and endocytic function are reduced by overexpression of the FE65 adaptor protein, FE65L1.

Guénette, Suzanne Y; Chang, Yang; Hyman, Bradley T; Tanzi, Rudolph E; Rebeck, G William.

J Neurochem ; 82(4): 755-62, 2002 Aug.

Artículo en Inglés | MEDLINE | ID: mdl-12358780

17.

Insulin-degrading enzyme regulates the levels of insulin, amyloid beta-protein, and the beta-amyloid precursor protein intracellular domain in vivo.

Farris, Wesley; Mansourian, Stefan; Chang, Yang; Lindsley, Loren; Eckman, Elizabeth A; Frosch, Matthew P; Eckman, Christopher B; Tanzi, Rudolph E; Selkoe, Dennis J; Guenette, Suzanne.

Proc Natl Acad Sci U S A ; 100(7): 4162-7, 2003 Apr 01.

Artículo en Inglés | MEDLINE | ID: mdl-12634421

18.

Generation of the beta-amyloid peptide and the amyloid precursor protein C-terminal fragment gamma are potentiated by FE65L1.

Chang, Yang; Tesco, Giuseppina; Jeong, William J; Lindsley, Loren; Eckman, Elizabeth A; Eckman, Christopher B; Tanzi, Rudolph E; Guénette, Suzanne Y.

J Biol Chem ; 278(51): 51100-7, 2003 Dec 19.

Artículo en Inglés | MEDLINE | ID: mdl-14527950

19.

APP at a glance.

Wolfe, Michael S; Guénette, Suzanne Y.

J Cell Sci ; 120(Pt 18): 3157-61, 2007 Sep 15.

Artículo en Inglés | MEDLINE | ID: mdl-17878232

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FE65 and FE65L1 amyloid precursor protein-binding protein compound null mice display adult-onset cataract and muscle weakness.

Amyloid beta a4 precursor protein-binding family B member 1 (FE65) interactomics revealed synaptic vesicle glycoprotein 2A (SV2A) and sarcoplasmic/endoplasmic reticulum calcium ATPase 2 (SERCA2) as new binding proteins in the human brain.

A role for FE65 in controlling GnRH-1 neurogenesis.

FE65 proteins regulate NMDA receptor activation-induced amyloid precursor protein processing.

Hypoxic ischemia and proteasome dysfunction alter tau isoform ratio by inhibiting exon 10 splicing.

APP Protein Family Signaling at the Synapse: Insights from Intracellular APP-Binding Proteins.

FE65 and FE65L1 share common synaptic functions and genetically interact with the APP family in neuromuscular junction formation.

A role for APP in motility and transcription?

Mechanisms of Abeta clearance and catabolism.

No major role for insulin-degrading enzyme in antigen presentation by MHC molecules.

FE65 as a link between VLDLR and APP to regulate their trafficking and processing.

Astrocytes: a cellular player in Abeta clearance and degradation.

HtrA2 regulates beta-amyloid precursor protein (APP) metabolism through endoplasmic reticulum-associated degradation.

FE65 interaction with the ApoE receptor ApoEr2.

Essential roles for the FE65 amyloid precursor protein-interacting proteins in brain development.

Low-density lipoprotein receptor-related protein levels and endocytic function are reduced by overexpression of the FE65 adaptor protein, FE65L1.

Insulin-degrading enzyme regulates the levels of insulin, amyloid beta-protein, and the beta-amyloid precursor protein intracellular domain in vivo.

Generation of the beta-amyloid peptide and the amyloid precursor protein C-terminal fragment gamma are potentiated by FE65L1.

APP at a glance.