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1.
Clinical performance of Rb-82 myocardial perfusion PET and Tc-99m-based SPECT in patients with extreme obesity.
J Nucl Cardiol
; 26(1): 275-283, 2019 02.
Artículo
en Inglés
| MEDLINE | ID: mdl-28357812
2.
Towards the discovery of drug-like epigallocatechin gallate analogs as Hsp90 inhibitors.
Bioorg Med Chem Lett
; 24(10): 2263-6, 2014 May 15.
Artículo
en Inglés
| MEDLINE | ID: mdl-24745965
3.
(-)-Epigallocatechin-3-gallate is a novel Hsp90 inhibitor.
Biochemistry
; 48(2): 336-45, 2009 Jan 20.
Artículo
en Inglés
| MEDLINE | ID: mdl-19113837
4.
Expression and activity of aryl hydrocarbon receptors in development and cancer.
Crit Rev Eukaryot Gene Expr
; 18(4): 279-321, 2008.
Artículo
en Inglés
| MEDLINE | ID: mdl-18652561
5.
Aryl Hydrocarbon Receptor Deficiency in an Exon 3 Deletion Mouse Model Promotes Hematopoietic Stem Cell Proliferation and Impacts Endosteal Niche Cells.
Stem Cells Int
; 2016: 4536187, 2016.
Artículo
en Inglés
| MEDLINE | ID: mdl-27366154
6.
The heat shock protein 90 inhibitor, (-)-epigallocatechin gallate, has anticancer activity in a novel human prostate cancer progression model.
Cancer Prev Res (Phila)
; 8(3): 249-57, 2015 Mar.
Artículo
en Inglés
| MEDLINE | ID: mdl-25604133
7.
Neural precursor cell proliferation is disrupted through activation of the aryl hydrocarbon receptor by 2,3,7,8-tetrachlorodibenzo-p-dioxin.
Stem Cells Dev
; 20(2): 313-26, 2011 Feb.
Artículo
en Inglés
| MEDLINE | ID: mdl-20486776
8.
TCDD and a putative endogenous AhR ligand, ITE, elicit the same immediate changes in gene expression in mouse lung fibroblasts.
Toxicol Sci
; 114(1): 90-100, 2010 Mar.
Artículo
en Inglés
| MEDLINE | ID: mdl-19933214
9.
Novel compound 2-methyl-2H-pyrazole-3-carboxylic acid (2-methyl-4-o-tolylazo-phenyl)-amide (CH-223191) prevents 2,3,7,8-TCDD-induced toxicity by antagonizing the aryl hydrocarbon receptor.
Mol Pharmacol
; 69(6): 1871-8, 2006 Jun.
Artículo
en Inglés
| MEDLINE | ID: mdl-16540597
10.
Species-specific transcriptional activity of synthetic flavonoids in guinea pig and mouse cells as a result of differential activation of the aryl hydrocarbon receptor to interact with dioxin-responsive elements.
Mol Pharmacol
; 63(4): 915-24, 2003 Apr.
Artículo
en Inglés
| MEDLINE | ID: mdl-12644593
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