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1.
Interaction between 17ß-estradiol, prolactin and human papillomavirus induce E6/E7 transcript and modulate the expression and localization of hormonal receptors.
Cancer Cell Int
; 19: 227, 2019.
Artículo
en Inglés
| MEDLINE | ID: mdl-31507337
2.
Hormonal modulation of Toxoplasma gondii infection: Regulation of hormonal receptors and cytokine production in THP-1 cells.
Exp Parasitol
; 204: 107721, 2019 Sep.
Artículo
en Inglés
| MEDLINE | ID: mdl-31288023
3.
STAT3 activation is required for the antiapoptotic effects of prolactin in cervical cancer cells.
Cancer Cell Int
; 15: 83, 2015.
Artículo
en Inglés
| MEDLINE | ID: mdl-26346346
4.
High expression of prolactin receptor is associated with cell survival in cervical cancer cells.
Cancer Cell Int
; 13(1): 103, 2013 Oct 22.
Artículo
en Inglés
| MEDLINE | ID: mdl-24148306
5.
17ß-estradiol modulates the expression of hormonal receptors on THP-1 T. gondii-infected macrophages and monocytes in an AKT and ERK-dependent manner.
Mol Biochem Parasitol
; 247: 111433, 2022 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-34822916
6.
GPER Overexpression in Cervical Cancer Versus Premalignant Lesions: Its Activation Induces Different Forms of Cell Death.
Anticancer Agents Med Chem
; 19(6): 783-791, 2019.
Artículo
en Inglés
| MEDLINE | ID: mdl-30727915
7.
A 60 kDa prolactin variant secreted by cervical cancer cells modulates apoptosis and cytokine production.
Oncol Rep
; 39(3): 1253-1260, 2018 Mar.
Artículo
en Inglés
| MEDLINE | ID: mdl-29399697
8.
Effects of 60 kDa prolactin and estradiol on metabolism and cell survival in cervical cancer: Coexpression of their hormonal receptors during cancer progression.
Oncol Rep
; 40(6): 3781-3793, 2018 Dec.
Artículo
en Inglés
| MEDLINE | ID: mdl-30272319
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