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1.
IL-1ß is a key inflammatory cytokine that weakens lactation-specific tight junctions of mammary epithelial cells.
Exp Cell Res
; 409(2): 112938, 2021 12 15.
Artículo
en Inglés
| MEDLINE | ID: mdl-34800541
2.
IL-1ß directly inhibits milk lipid production in lactating mammary epithelial cells concurrently with enlargement of cytoplasmic lipid droplets.
Exp Cell Res
; 370(2): 365-372, 2018 09 15.
Artículo
en Inglés
| MEDLINE | ID: mdl-29966663
3.
Moderate High Temperature Condition Induces the Lactation Capacity of Mammary Epithelial Cells Through Control of STAT3 and STAT5 Signaling.
J Mammary Gland Biol Neoplasia
; 23(1-2): 75-88, 2018 06.
Artículo
en Inglés
| MEDLINE | ID: mdl-29633073
4.
Prolactin and glucocorticoid signaling induces lactation-specific tight junctions concurrent with ß-casein expression in mammary epithelial cells.
Biochim Biophys Acta
; 1863(8): 2006-16, 2016 08.
Artículo
en Inglés
| MEDLINE | ID: mdl-27130254
5.
Phytoestrogens Weaken the Blood-Milk Barrier in Lactating Mammary Epithelial Cells by Affecting Tight Junctions and Cell Viability.
J Agric Food Chem
; 65(50): 11118-11124, 2017 Dec 20.
Artículo
en Inglés
| MEDLINE | ID: mdl-29189005
6.
Isoflavones and their metabolites influence the milk component synthesis ability of mammary epithelial cells through prolactin/STAT5 signaling.
Mol Nutr Food Res
; 61(10)2017 10.
Artículo
en Inglés
| MEDLINE | ID: mdl-28605125
7.
Distinct roles of prolactin, epidermal growth factor, and glucocorticoids in ß-casein secretion pathway in lactating mammary epithelial cells.
Mol Cell Endocrinol
; 440: 16-24, 2017 01 15.
Artículo
en Inglés
| MEDLINE | ID: mdl-27836773
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