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1.
Barth syndrome: cellular compensation of mitochondrial dysfunction and apoptosis inhibition due to changes in cardiolipin remodeling linked to tafazzin (TAZ) gene mutation.
Biochim Biophys Acta
; 1832(8): 1194-206, 2013 Aug.
Artículo
en Inglés
| MEDLINE | ID: mdl-23523468
2.
Synthesis of new flavonoid derivatives based on 3-hydroxy-4'-dimethylamino flavone and study the activity of some of them as antifungal.
Heliyon
; 8(12): e12062, 2022 Dec.
Artículo
en Inglés
| MEDLINE | ID: mdl-36561670
3.
Curcumin, a Multifaceted Hormetic Agent, Mediates an Intricate Crosstalk between Mitochondrial Turnover, Autophagy, and Apoptosis.
Oxid Med Cell Longev
; 2020: 3656419, 2020.
Artículo
en Inglés
| MEDLINE | ID: mdl-32765806
4.
Highlighting Curcumin-Induced Crosstalk between Autophagy and Apoptosis as Supported by Its Specific Subcellular Localization.
Cells
; 9(2)2020 02 04.
Artículo
en Inglés
| MEDLINE | ID: mdl-32033136
5.
Iron chelation by curcumin suppresses both curcumin-induced autophagy and cell death together with iron overload neoplastic transformation.
Cell Death Discov
; 5: 150, 2019.
Artículo
en Inglés
| MEDLINE | ID: mdl-31839992
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