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1.
Loss of positive allosteric interactions between neuronal nitric oxide synthase and phosphofructokinase contributes to defects in glycolysis and increased fatigability in muscular dystrophy.
Hum Mol Genet
; 18(18): 3439-51, 2009 Sep 15.
Artículo
en Inglés
| MEDLINE | ID: mdl-19542095
2.
Major basic protein-1 promotes fibrosis of dystrophic muscle and attenuates the cellular immune response in muscular dystrophy.
Hum Mol Genet
; 17(15): 2280-92, 2008 Aug 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-18430716
3.
Age-related loss of nitric oxide synthase in skeletal muscle causes reductions in calpain S-nitrosylation that increase myofibril degradation and sarcopenia.
Aging Cell
; 11(6): 1036-45, 2012 Dec.
Artículo
en Inglés
| MEDLINE | ID: mdl-22950758
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