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1.
Toll-like receptor 2, hyaluronan, and neutrophils play a key role in plaque erosion: the OPTICO-ACS study.
Eur Heart J
; 44(38): 3892-3907, 2023 10 12.
Artículo
en Inglés
| MEDLINE | ID: mdl-37381760
2.
Culprit plaque morphology determines inflammatory risk and clinical outcomes in acute coronary syndrome.
Eur Heart J
; 44(38): 3911-3925, 2023 10 12.
Artículo
en Inglés
| MEDLINE | ID: mdl-37381774
3.
Coronary microevaginations characterize culprit plaques and their inflammatory microenvironment in a subtype of acute coronary syndrome with intact fibrous cap: results from the prospective translational OPTICO-ACS study.
Eur Heart J Cardiovasc Imaging
; 25(2): 175-184, 2024 Jan 29.
Artículo
en Inglés
| MEDLINE | ID: mdl-37395586
4.
Cholesterol crystals at the culprit lesion in patients with acute coronary syndrome are associated with worse cardiovascular outcomes at two years follow up - results from the translational OPTICO-ACS study program.
Int J Cardiol
; 399: 131665, 2024 Mar 15.
Artículo
en Inglés
| MEDLINE | ID: mdl-38141724
5.
Spotty calcium deposits within acute coronary syndrome (ACS)-causing culprit lesions impact inflammatory vessel-wall interactions and are associated with higher cardiovascular event rates at one year follow-up: Results from the prospective translational OPTICO-ACS study program.
Atherosclerosis
; 385: 117284, 2023 11.
Artículo
en Inglés
| MEDLINE | ID: mdl-37871405
6.
Coexistence of calcified- and lipid-containing plaque components and their association with incidental rupture points in acute coronary syndrome-causing culprit lesions: results from the prospective OPTICO-ACS study.
Eur Heart J Cardiovasc Imaging
; 23(12): 1598-1605, 2022 11 17.
Artículo
en Inglés
| MEDLINE | ID: mdl-34904655
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