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1.
Reduced NODAL signaling strength via mutation of several pathway members including FOXH1 is linked to human heart defects and holoprosencephaly.
Am J Hum Genet
; 83(1): 18-29, 2008 Jul.
Artículo
en Inglés
| MEDLINE | ID: mdl-18538293
2.
Truncating loss-of-function mutations of DISP1 contribute to holoprosencephaly-like microform features in humans.
Hum Genet
; 125(4): 393-400, 2009 May.
Artículo
en Inglés
| MEDLINE | ID: mdl-19184110
3.
Cumulative ligand activity of NODAL mutations and modifiers are linked to human heart defects and holoprosencephaly.
Mol Genet Metab
; 98(1-2): 225-34, 2009.
Artículo
en Inglés
| MEDLINE | ID: mdl-19553149
4.
Risk classification at the time of diagnosis differentially affects the level of residual disease in children with B-precursor acute lymphoblastic leukemia after completion of therapy.
Leuk Res
; 27(8): 743-50, 2003 Aug.
Artículo
en Inglés
| MEDLINE | ID: mdl-12801533
5.
An early requirement for maternal FoxH1 during zebrafish gastrulation.
Dev Biol
; 310(1): 10-22, 2007 Oct 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-17719025
6.
Residual leukaemia after bone marrow transplant in children with acute lymphoblastic leukaemia after first haematological relapse or with poor initial presenting features.
Br J Haematol
; 120(4): 711-5, 2003 Feb.
Artículo
en Inglés
| MEDLINE | ID: mdl-12588362
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