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1.
Circulating clinical strains of human parainfluenza virus reveal viral entry requirements for in vivo infection.
J Virol
; 88(22): 13495-502, 2014 Nov.
Artículo
en Inglés
| MEDLINE | ID: mdl-25210187
2.
Identification of a region in the stalk domain of the nipah virus receptor binding protein that is critical for fusion activation.
J Virol
; 87(20): 10980-96, 2013 Oct.
Artículo
en Inglés
| MEDLINE | ID: mdl-23903846
3.
Mechanism of fusion triggering by human parainfluenza virus type III: communication between viral glycoproteins during entry.
J Biol Chem
; 287(1): 778-793, 2012 Jan 02.
Artículo
en Inglés
| MEDLINE | ID: mdl-22110138
4.
The second receptor binding site of the globular head of the Newcastle disease virus hemagglutinin-neuraminidase activates the stalk of multiple paramyxovirus receptor binding proteins to trigger fusion.
J Virol
; 86(10): 5730-41, 2012 May.
Artículo
en Inglés
| MEDLINE | ID: mdl-22438532
5.
Spring-loaded model revisited: paramyxovirus fusion requires engagement of a receptor binding protein beyond initial triggering of the fusion protein.
J Virol
; 85(24): 12867-80, 2011 Dec.
Artículo
en Inglés
| MEDLINE | ID: mdl-21976650
6.
Human parainfluenza virus infection of the airway epithelium: viral hemagglutinin-neuraminidase regulates fusion protein activation and modulates infectivity.
J Virol
; 83(13): 6900-8, 2009 Jul.
Artículo
en Inglés
| MEDLINE | ID: mdl-19386708
7.
Interaction between the hemagglutinin-neuraminidase and fusion glycoproteins of human parainfluenza virus type III regulates viral growth in vivo.
mBio
; 4(5): e00803-13, 2013 Oct 22.
Artículo
en Inglés
| MEDLINE | ID: mdl-24149514
8.
Adaptation of human parainfluenza virus to airway epithelium reveals fusion properties required for growth in host tissue.
mBio
; 3(3)2012.
Artículo
en Inglés
| MEDLINE | ID: mdl-22669629
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