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1.
Critical role of airway macrophages in modulating disease severity during influenza virus infection of mice.
J Virol
; 84(15): 7569-80, 2010 Aug.
Artículo
en Inglés
| MEDLINE | ID: mdl-20504924
2.
Depletion of Gr-1+, but not Ly6G+, immune cells exacerbates virus replication and disease in an intranasal model of herpes simplex virus type 1 infection.
J Gen Virol
; 91(Pt 9): 2158-66, 2010 Sep.
Artículo
en Inglés
| MEDLINE | ID: mdl-20538903
3.
Influenza viruses differ in ability to infect macrophages and to induce a local inflammatory response following intraperitoneal injection of mice.
Immunol Cell Biol
; 88(6): 641-50, 2010 Aug.
Artículo
en Inglés
| MEDLINE | ID: mdl-20142836
4.
Loss of a single N-linked glycan from the hemagglutinin of influenza virus is associated with resistance to collectins and increased virulence in mice.
Respir Res
; 10: 117, 2009 Nov 23.
Artículo
en Inglés
| MEDLINE | ID: mdl-19930664
5.
Glycosylation as a target for recognition of influenza viruses by the innate immune system.
Adv Exp Med Biol
; 598: 279-92, 2007.
Artículo
en Inglés
| MEDLINE | ID: mdl-17892219
6.
Gr-1+ cells, but not neutrophils, limit virus replication and lesion development following flank infection of mice with herpes simplex virus type-1.
Virology
; 407(1): 143-51, 2010 Nov 10.
Artículo
en Inglés
| MEDLINE | ID: mdl-20817252
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