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1.
Studies on the role of apoptosis after transient myocardial ischemia: genetic deletion of the executioner caspases-3 and -7 does not limit infarct size and ventricular remodeling.
Basic Res Cardiol
; 111(2): 18, 2016 Mar.
Artículo
en Inglés
| MEDLINE | ID: mdl-26924441
2.
cGMP/PKG pathway mediates myocardial postconditioning protection in rat hearts by delaying normalization of intracellular acidosis during reperfusion.
J Mol Cell Cardiol
; 50(5): 903-9, 2011 May.
Artículo
en Inglés
| MEDLINE | ID: mdl-21362429
3.
Calpain translocation and activation as pharmacological targets during myocardial ischemia/reperfusion.
J Mol Cell Cardiol
; 49(2): 271-9, 2010 Aug.
Artículo
en Inglés
| MEDLINE | ID: mdl-20211186
4.
Delayed phospholamban phosphorylation in post-conditioned heart favours Ca2+ normalization and contributes to protection.
Cardiovasc Res
; 103(4): 542-53, 2014 Sep 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-25020913
5.
Activation of cGMP/protein kinase G pathway in postconditioned myocardium depends on reduced oxidative stress and preserved endothelial nitric oxide synthase coupling.
J Am Heart Assoc
; 2(1): e005975, 2013 Jan 02.
Artículo
en Inglés
| MEDLINE | ID: mdl-23525447
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