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1.
Acetylcholinesterase inhibition protects against trastuzumab-induced cardiotoxicity through reducing multiple programmed cell death pathways.
Mol Med
; 29(1): 123, 2023 09 11.
Artículo
en Inglés
| MEDLINE | ID: mdl-37691124
2.
Vagus nerve stimulation exerts cardioprotection against doxorubicin-induced cardiotoxicity through inhibition of programmed cell death pathways.
Cell Mol Life Sci
; 80(1): 21, 2022 Dec 30.
Artículo
en Inglés
| MEDLINE | ID: mdl-36583785
3.
Promoting mitochondrial fusion in doxorubicin-induced cardiotoxicity: a novel therapeutic target for cardioprotection.
Clin Sci (Lond)
; 136(11): 841-860, 2022 06 17.
Artículo
en Inglés
| MEDLINE | ID: mdl-35543245
4.
Ranolazine alleviated cardiac/brain dysfunction in doxorubicin-treated rats.
Exp Mol Pathol
; 127: 104818, 2022 08.
Artículo
en Inglés
| MEDLINE | ID: mdl-35882281
5.
Extracellular Vesicles Released after Doxorubicin Treatment in Rats Protect Cardiomyocytes from Oxidative Damage and Induce Pro-Inflammatory Gene Expression in Macrophages.
Int J Mol Sci
; 23(21)2022 Nov 03.
Artículo
en Inglés
| MEDLINE | ID: mdl-36362245
6.
Acetylcholinesterase inhibitor ameliorates doxorubicin-induced cardiotoxicity through reducing RIP1-mediated necroptosis.
Pharmacol Res
; 173: 105882, 2021 11.
Artículo
en Inglés
| MEDLINE | ID: mdl-34530122
7.
Corrigendum to "Acetylcholinesterase inhibitor ameliorates doxorubicin-induced cardiotoxicity through reducing RIP1-mediated necroptosis" [Pharmacol. Res. 173 (2021) 105882].
Pharmacol Res
; 203: 107147, 2024 May.
Artículo
en Inglés
| MEDLINE | ID: mdl-38565498
8.
Nicotinic and Muscarinic Acetylcholine Receptor Agonists Counteract Cognitive Impairment in a Rat Model of Doxorubicin-Induced Chemobrain via Attenuation of Multiple Programmed Cell Death Pathways.
Mol Neurobiol
; 2024 Apr 03.
Artículo
en Inglés
| MEDLINE | ID: mdl-38568417
9.
Melatonin and metformin counteract cognitive dysfunction equally in male rats with doxorubicin-induced chemobrain.
Neurotoxicology
; 94: 158-171, 2023 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-36463981
10.
Melatonin and metformin ameliorated trastuzumab-induced cardiotoxicity through the modulation of mitochondrial function and dynamics without reducing its anticancer efficacy.
Biochim Biophys Acta Mol Basis Dis
; 1869(2): 166618, 2023 02.
Artículo
en Inglés
| MEDLINE | ID: mdl-36494039
11.
Potential roles of 4HNE-adducted protein in serum extracellular vesicles as an early indicator of oxidative response against doxorubicin-induced cardiomyopathy in rats.
Curr Res Toxicol
; 5: 100134, 2023.
Artículo
en Inglés
| MEDLINE | ID: mdl-37964944
12.
Vagus nerve stimulation and acetylcholinesterase inhibitor donepezil provide cardioprotection against trastuzumab-induced cardiotoxicity in rats by attenuating mitochondrial dysfunction.
Biochem Pharmacol
; 217: 115836, 2023 11.
Artículo
en Inglés
| MEDLINE | ID: mdl-37816466
13.
Acetylcholine receptor agonists effectively attenuated multiple program cell death pathways and improved left ventricular function in trastuzumab-induced cardiotoxicity in rats.
Life Sci
; 329: 121971, 2023 Sep 15.
Artículo
en Inglés
| MEDLINE | ID: mdl-37482212
14.
Acetylcholine receptor agonists provide cardioprotection in doxorubicin-induced cardiotoxicity via modulating muscarinic M2 and α7 nicotinic receptor expression.
Transl Res
; 243: 33-51, 2022 05.
Artículo
en Inglés
| MEDLINE | ID: mdl-34920165
15.
Cardioprotective effects of melatonin and metformin against doxorubicin-induced cardiotoxicity in rats are through preserving mitochondrial function and dynamics.
Biochem Pharmacol
; 192: 114743, 2021 10.
Artículo
en Inglés
| MEDLINE | ID: mdl-34453902
16.
Effects of doxorubicin on the heart: From molecular mechanisms to intervention strategies.
Eur J Pharmacol
; 866: 172818, 2020 Jan 05.
Artículo
en Inglés
| MEDLINE | ID: mdl-31758940
17.
Corrigendum to "Cardioprotective effects of melatonin and metformin against doxorubicin-induced cardiotoxicity in rats are through preserving mitochondrial function and dynamics" [Biochem. Pharmacol. 192 (2021) 114743].
Biochem Pharmacol
; 208: 115414, 2023 Feb.
Artículo
en Inglés
| MEDLINE | ID: mdl-36680972
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