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1.
Loss of Toll-like receptor 2 and 4 leads to differential induction of endoplasmic reticulum stress and proapoptotic responses in the intestinal epithelium under conditions of chronic inflammation.
J Proteome Res
; 8(10): 4406-17, 2009 Oct.
Artículo
en Inglés
| MEDLINE | ID: mdl-19681597
2.
rs224136 on chromosome 10q21.1 and variants in PHOX2B, NCF4, and FAM92B are not major genetic risk factors for susceptibility to Crohn's disease in the German population.
Am J Gastroenterol
; 104(3): 665-72, 2009 Mar.
Artículo
en Inglés
| MEDLINE | ID: mdl-19262523
3.
Linking genetic susceptibility to Crohn's disease with Th17 cell function: IL-22 serum levels are increased in Crohn's disease and correlate with disease activity and IL23R genotype status.
Inflamm Bowel Dis
; 14(2): 204-12, 2008 Feb.
Artículo
en Inglés
| MEDLINE | ID: mdl-18022867
4.
Role of the novel Th17 cytokine IL-17F in inflammatory bowel disease (IBD): upregulated colonic IL-17F expression in active Crohn's disease and analysis of the IL17F p.His161Arg polymorphism in IBD.
Inflamm Bowel Dis
; 14(4): 437-45, 2008 Apr.
Artículo
en Inglés
| MEDLINE | ID: mdl-18088064
5.
The ATG16L1 gene variants rs2241879 and rs2241880 (T300A) are strongly associated with susceptibility to Crohn's disease in the German population.
Am J Gastroenterol
; 103(3): 682-91, 2008 Mar.
Artículo
en Inglés
| MEDLINE | ID: mdl-18162085
6.
rs1004819 is the main disease-associated IL23R variant in German Crohn's disease patients: combined analysis of IL23R, CARD15, and OCTN1/2 variants.
PLoS One
; 2(9): e819, 2007 Sep 05.
Artículo
en Inglés
| MEDLINE | ID: mdl-17786191
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