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1.
B2 cells contribute to hypertension and natural killer cell activation possibly via AT1-AA in response to placental ischemia.
Am J Physiol Renal Physiol
; 324(2): F179-F192, 2023 02 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-36417275
2.
AT1-AA Is Produced in Offspring in Response to Placental Ischemia and Is Lowered by B-Cell Depletion Without Compromising Overall Offspring Health.
J Am Heart Assoc
; 13(4): e031417, 2024 Feb 20.
Artículo
en Inglés
| MEDLINE | ID: mdl-38353227
3.
The role of T cell stimulated agonistic autoantibodies to the angiotensin II type I receptor (AT1-AA) in mediating multiorgan dysfunction in IL-17 induced hypertension during pregnancy.
Am J Reprod Immunol
; 91(4): e13843, 2024 Apr.
Artículo
en Inglés
| MEDLINE | ID: mdl-38606700
4.
AT1-AA Infusion during Pregnancy Impairs CBF Autoregulation Postpartum.
Int J Cerebrovasc Dis Stroke
; 6(1)2023.
Artículo
en Inglés
| MEDLINE | ID: mdl-37901747
5.
RUPP Th17s cause hypertension and mitochondrial dysfunction in the kidney and placenta during pregnancy.
Pregnancy Hypertens
; 32: 50-56, 2023 Jun.
Artículo
en Inglés
| MEDLINE | ID: mdl-37104924
6.
Inhibiting B cell activating factor attenuates preeclamptic symptoms in placental ischemic rats.
Am J Reprod Immunol
; 89(4): e13693, 2023 04.
Artículo
en Inglés
| MEDLINE | ID: mdl-36794639
7.
Inhibition of angiotensin II type 1 receptor agonistic autoantibodies by direct binding does not impact reduced uterine perfusion pressure offspring birthweight and blood pressure at adulthood.
Am J Obstet Gynecol MFM
; 5(6): 100945, 2023 06.
Artículo
en Inglés
| MEDLINE | ID: mdl-36990181
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