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1.
NF-κB activation mediates LPS-or zymosan-induced hypotension and inflammation reversed by BAY61-3606, a selective Syk inhibitor, in rat models of septic and non-septic shock.
Clin Exp Pharmacol Physiol
; 46(2): 173-182, 2019 02.
Artículo
en Inglés
| MEDLINE | ID: mdl-30347478
2.
The role of Syk/IĸB-α/NF-ĸB pathway activation in the reversal effect of BAY 61-3606, a selective Syk inhibitor, on hypotension and inflammation in a rat model of zymosan-induced non-septic shock.
Clin Exp Pharmacol Physiol
; 45(2): 155-165, 2018 02.
Artículo
en Inglés
| MEDLINE | ID: mdl-28949404
3.
Effects of 5,14-HEDGE, a 20-HETE mimetic, on lipopolysaccharide-induced changes in MyD88/TAK1/IKKß/IκB-α/NF-κB pathway and circulating miR-150, miR-223, and miR-297 levels in a rat model of septic shock.
Inflamm Res
; 63(9): 741-56, 2014 Sep.
Artículo
en Inglés
| MEDLINE | ID: mdl-24915805
4.
Contribution of iNOS/sGC/PKG pathway, COX-2, CYP4A1, and gp91(phox) to the protective effect of 5,14-HEDGE, a 20-HETE mimetic, against vasodilation, hypotension, tachycardia, and inflammation in a rat model of septic shock.
Nitric Oxide
; 33: 18-41, 2013 Sep 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-23684565
5.
NS-398 reverses hypotension in endotoxemic rats: contribution of eicosanoids, NO, and peroxynitrite.
Prostaglandins Other Lipid Mediat
; 104-105: 93-108, 2013.
Artículo
en Inglés
| MEDLINE | ID: mdl-22975359
6.
5,14-HEDGE, a 20-HETE mimetic, reverses hypotension and improves survival in a rodent model of septic shock: contribution of soluble epoxide hydrolase, CYP2C23, MEK1/ERK1/2/IKKß/IκB-α/NF-κB pathway, and proinflammatory cytokine formation.
Prostaglandins Other Lipid Mediat
; 102-103: 31-41, 2013.
Artículo
en Inglés
| MEDLINE | ID: mdl-23454652
7.
Contribution of RhoA/Rho-kinase/MEK1/ERK1/2/iNOS pathway to ischemia/reperfusion-induced oxidative/nitrosative stress and inflammation leading to distant and target organ injury in rats.
Eur J Pharmacol
; 723: 234-45, 2014 Jan 15.
Artículo
en Inglés
| MEDLINE | ID: mdl-24296316
8.
Increased production of nitric oxide mediates selective organ-specific effects of endotoxin on oxidative stress.
Antiinflamm Antiallergy Agents Med Chem
; 11(2): 161-72, 2012.
Artículo
en Inglés
| MEDLINE | ID: mdl-23173576
9.
A novel treatment strategy for sepsis and septic shock based on the interactions between prostanoids, nitric oxide, and 20-hydroxyeicosatetraenoic acid.
Antiinflamm Antiallergy Agents Med Chem
; 11(2): 121-50, 2012.
Artículo
en Inglés
| MEDLINE | ID: mdl-23013331
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