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1.
c-Jun signaling during initial HSV-1 infection modulates latency to enhance later reactivation in addition to directly promoting the progression to full reactivation.
J Virol
; 98(2): e0176423, 2024 Feb 20.
Artículo
en Inglés
| MEDLINE | ID: mdl-38193709
2.
HSV-1 miRNAs are post-transcriptionally edited in latently infected human ganglia.
J Virol
; 97(10): e0073023, 2023 10 31.
Artículo
en Inglés
| MEDLINE | ID: mdl-37712701
3.
Ex Vivo Herpes Simplex Virus Reactivation Involves a Dual Leucine Zipper Kinase-Dependent Wave of Lytic Gene Expression That Is Independent of Histone Demethylase Activity and Viral Genome Synthesis.
J Virol
; 96(12): e0047522, 2022 06 22.
Artículo
en Inglés
| MEDLINE | ID: mdl-35604215
4.
Key questions on the epigenetics of herpes simplex virus latency.
PLoS Pathog
; 18(6): e1010587, 2022 06.
Artículo
en Inglés
| MEDLINE | ID: mdl-35772067
5.
Single-genome analysis reveals a heterogeneous association of the herpes simplex virus genome with H3K27me2 and the reader PHF20L1 following infection of human fibroblasts.
mBio
; 15(4): e0327823, 2024 Apr 10.
Artículo
en Inglés
| MEDLINE | ID: mdl-38411116
6.
c-Jun Signaling During Initial HSV-1 Infection Modulates Latency to Enhance Later Reactivation in addition to Directly Promoting the Progression to Full Reactivation.
bioRxiv
; 2023 Nov 10.
Artículo
en Inglés
| MEDLINE | ID: mdl-37986840
7.
Single-genome analysis reveals heterogeneous association of the Herpes Simplex Virus genome with H3K27me2 and the reader PHF20L1 following infection of human fibroblasts.
bioRxiv
; 2023 Dec 03.
Artículo
en Inglés
| MEDLINE | ID: mdl-38076966
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