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1.
ROS- and HIF1α-dependent IGFBP3 upregulation blocks IGF1 survival signaling and thereby mediates high-glucose-induced cardiomyocyte apoptosis.
J Cell Physiol
; 234(8): 13557-13570, 2019 08.
Artículo
en Inglés
| MEDLINE | ID: mdl-30659610
2.
Diallyl trisulfide suppresses doxorubicin-induced cardiomyocyte apoptosis by inhibiting MAPK/NF-κB signaling through attenuation of ROS generation.
Environ Toxicol
; 33(1): 93-103, 2018 Jan.
Artículo
en Inglés
| MEDLINE | ID: mdl-29087013
3.
Nrf2 Activation as a Protective Feedback to Limit Cell Death in High Glucose-Exposed Cardiomyocytes.
J Cell Biochem
; 118(7): 1659-1669, 2017 07.
Artículo
en Inglés
| MEDLINE | ID: mdl-27859591
4.
Lactate dehydrogenase downregulation mediates the inhibitory effect of diallyl trisulfide on proliferation, metastasis, and invasion in triple-negative breast cancer.
Environ Toxicol
; 32(4): 1390-1398, 2017 Apr.
Artículo
en Inglés
| MEDLINE | ID: mdl-27566995
5.
Pkcδ Activation is Involved in ROS-Mediated Mitochondrial Dysfunction and Apoptosis in Cardiomyocytes Exposed to Advanced Glycation End Products (Ages).
Aging Dis
; 9(4): 647-663, 2018 Aug.
Artículo
en Inglés
| MEDLINE | ID: mdl-30090653
6.
Diallyl trisulfide protects against high glucose-induced cardiac apoptosis by stimulating the production of cystathionine gamma-lyase-derived hydrogen sulfide.
Int J Cardiol
; 195: 300-10, 2015 Sep 15.
Artículo
en Inglés
| MEDLINE | ID: mdl-26056963
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