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1.
A dominant-negative mutation of the growth hormone receptor causes familial short stature.
Nat Genet
; 16(1): 13-4, 1997 May.
Artículo
en Inglés
| MEDLINE | ID: mdl-9140387
2.
A short isoform of the human growth hormone receptor functions as a dominant negative inhibitor of the full-length receptor and generates large amounts of binding protein.
Mol Endocrinol
; 11(3): 265-73, 1997 Mar.
Artículo
en Inglés
| MEDLINE | ID: mdl-9058373
3.
High-dose growth hormone does not affect proinflammatory cytokine (tumor necrosis factor-alpha, interleukin-6, and interferon-gamma) release from activated peripheral blood mononuclear cells or after minimal to moderate surgical stress.
J Clin Endocrinol Metab
; 85(9): 3383-90, 2000 Sep.
Artículo
en Inglés
| MEDLINE | ID: mdl-10999838
4.
Stimulation of endogenous GH and interleukin-6 receptors selectively activates different Jaks and Stats, with a Stat5 specific synergistic effect of dexamethasone.
J Endocrinol
; 165(2): 301-11, 2000 May.
Artículo
en Inglés
| MEDLINE | ID: mdl-10810294
5.
Inflammatory cytokines and acquired growth hormone resistance.
Growth Horm IGF Res
; 10 Suppl B: S9-14, 2000 Apr.
Artículo
en Inglés
| MEDLINE | ID: mdl-10984247
6.
New growth hormone receptor exon 9 mutation causes genetic short stature.
Acta Paediatr Suppl
; 88(428): 168-72; discussion 173, 1999 Feb.
Artículo
en Inglés
| MEDLINE | ID: mdl-10102075
7.
Studies with a growth hormone antagonist and dual-fluorescent confocal microscopy demonstrate that the full-length human growth hormone receptor, but not the truncated isoform, is very rapidly internalized independent of Jak2-Stat5 signaling.
J Biol Chem
; 274(21): 14791-8, 1999 May 21.
Artículo
en Inglés
| MEDLINE | ID: mdl-10329677
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