Fragmentación del QRS y muerte súbita en paciente con displasia arritmogénica del ventrículo derecho

Rodríguez López, Luis A; Rodríguez Moreno, Eliany; Cruz Elizundia, Juan M; Díaz Sardiñas, Yohan M; Gavilanes Hernández, Reinaldo; Quenta Tarqui, Ruben R; González Achacollo, Jhosely A; Santana Santana, Carlos.
CorSalud ; 11(1): 70-74, ene.-mar. 2019. graf
Artículo en Español | LILACS | ID: biblio-1089712

Documentos relacionados

  1. Revisiting the Newly Modified Criteria for Arrhythmogenic Right Ventricular Cardiomyopathy/Dysplasia (ARVC/D) and Reporting Newly Identified Genes.
  2. The rare cause of ST segment elevation in left precordial leads - Diagnostic clues from subtle waveforms.
  3. Using Zebrafish Animal Model to Study the Genetic Underpinning and Mechanism of Arrhythmogenic Cardiomyopathy.
  4. Repolarization abnormalities unmasked with a 252-lead BSM system in patients with ARVC and healthy gene carriers.
  5. Chronological T-wave alternation before and after the onset of arrhythmogenic right ventricular cardiomyopathy.
  6. Desmoglein-2 mutations in propeptide cleavage-site causes arrhythmogenic right ventricular cardiomyopathy/dysplasia by impairing extracellular 1-dependent desmosomal interactions upon cellular stress.
  7. GCN5 contributes to intracellular lipid accumulation in human primary cardiac stromal cells from patients affected by Arrhythmogenic cardiomyopathy.
  8. Arrhythmogenic right ventricular cardiomyopathy : Evolving from unique clinical features to a complex pathophysiological concept.
  9. Desmosomal protein structure and function and the impact of disease-causing mutations.
  10. Cardiac desmosomal reserve: another piece of the exercise-induced arrhythmogenic cardiomyopathy puzzle?