Evaluation of energy metabolism in septic patients with and without AKI

Sanches, Ana Cláudia Soncini; Góes, Cassiana Regina de; Bufarah, Marina Nogueira Berbel; Oliveira, Mariana Cassani de; Balbi, André Luis; Ponce, Daniela.
Nutrire Rev. Soc. Bras. Aliment. Nutr ; 42: 1-6, Dec. 2017. tab
Artículo en Inglés | LILACS | ID: biblio-881213

Documentos relacionados

  1. The kidney protects against sepsis by producing systemic uromodulin.
  2. Erythropoietin promotes energy metabolism to improve LPS-induced injury in HK-2 cells via SIRT1/PGC1-α pathway.
  3. Renal mitochondrial dysfunction in ovine experimental sepsis-associated acute kidney injury.
  4. Involvement of Innate Immune System in the Pathogenesis of Sepsis-Associated Acute Kidney Injury.
  5. How Does Continuous Renal Replacement Therapy Affect Septic Acute Kidney Injury?
  6. Fibroblastic reticular cell-derived exosomes are a promising therapeutic approach for septic acute kidney injury.
  7. PHLDA1 knockdown inhibits inflammation and oxidative stress by regulating JNK/ERK pathway, and plays a protective role in sepsis-induced acute kidney injury.
  8. PARK7 is induced to protect against endotoxic acute kidney injury by suppressing NF-κB.
  9. Attenuation of Sepsis-Induced Acute Kidney Injury by Exogenous H<sub>2</sub>S via Inhibition of Ferroptosis.
  10. Evolution of altered tubular metabolism and mitochondrial function in sepsis-associated acute kidney injury.