Phylogenetic signal at the Cytb-SertRNA-IG1-ND1 mitochondrial region in Anopheles (Kerteszia) neivai Howard, Dyar & Knab, 1913

López-Rubio, Andrés; Suaza, Juan David; Porter, Charles; Uribe, Sandra; Bedoya, Gabriel; Vélez, Iván Darío.
Biomédica (Bogotá) ; 37(supl.2): 143-154, jul.-set. 2017. tab, graf
Artículo en Inglés | LILACS | ID: biblio-888533

Documentos relacionados

  1. [Phylogenetic signal at the Cytb-SertRNA-IG1-ND1 mitochondrial region in Anopheles (Kerteszia) neivai Howard, Dyar & Knab, 1913].
  2. Mitochondrial tRNA<sup>Ser(UCN)</sup> 7471delC may be a novel mutation associated with maternally transmitted hypertension.
  3. Mitochondrial genetic differentiation across populations of the malaria vector Anopheles lesteri from China (Diptera: Culicidae).
  4. Contribution of a mitochondrial tyrosyl-tRNA synthetase mutation to the phenotypic expression of the deafness-associated tRNA<sup>Ser(UCN)</sup> 7511A>G mutation.
  5. Novel mitochondrial DNA mutations responsible for maternally inherited nonsyndromic hearing loss.
  6. Haplogroup analysis supports a pathogenic role for the 7510T>C mutation of mitochondrial tRNA(Ser(UCN)) in sensorineural hearing loss.
  7. Fatal neonatal lactic acidosis caused by a novel de novo mitochondrial G7453A tRNA-Serine ((UCN)) mutation.
  8. Insights into substrate promiscuity of human seryl-tRNA synthetase.
  9. [Mutations of mitochondrial tRNA<sup>Ser(UCN)</sup> and their connection with hearing loss].
  10. Selective degradation of tRNASer(AGY) is the primary driver for mitochondrial seryl-tRNA synthetase-related disease.