Mice with genetic and induced B-cell deficiency as a model for disseminated encephalitozoonosis

Moysés, Carla Renata Serantoni; Alvares-Saraiva, Anuska Marcelino; Perez, Elizabeth Cristina; Spadacci-Morena, Diva Denelle; Costa, Lidiana Flora Vidôto da; Xavier, José Guilherme; Lallo, Maria Anete.
Comp Immunol Microbiol Infect Dis, v. 81, 101742, fev. 2022
Artículo en Inglés | SES-SP, SESSP-IBPROD, SES-SP | ID: bud-4106

Documentos relacionados

  1. Mice with genetic and induced B-cell deficiency as a model for disseminated encephalitozoonosis.
  2. Characterization of humoral and cell-mediated immunity in rabbits orally infected with Encephalitozoon cuniculi.
  3. B-1 cells upregulate CD8 T lymphocytes and increase proinflammatory cytokines serum levels in oral encephalitozoonosis.
  4. B-1 cell decreases susceptibility to encephalitozoonosis in mice.
  5. Opportunistic pneumonia caused by E. cuniculi in mice immunosuppressed with cyclophosphamide.
  6. A multidisciplinary review about Encephalitozoon cuniculi in a One Health perspective.
  7. Diabetes mellitus increases the susceptibility to encephalitozoonosis in mice.
  8. CD8 T lymphocytes from B-1 cell-deficient mice down-regulates fungicidal activity of macrophages challenged with E. Cuniculi.
  9. Raw Goat's Milk, Fresh and Soft Cheeses as a Potential Source of <i>Encephalitozoon cuniculi</i>.
  10. Microsporidia (Encephalitozoon cuniculi) in Patients with Degenerative Hip and Knee Disease, Czech Republic.