TNF-Ó y su receptor soluble en LES: implicaciones clínicas
Bezanilla L., María Antonieta; Guzmán Meléndez, María Antonieta; Agar Muñoz, Ana María.
Reumatología (Santiago de Chile)
; 11(1): 28-30, 1995. ilus, tab
Artículo en Español | LILACS | ID: lil-153027
Documentos relacionados
Structural modeling of tumor necrosis factor: A protein of immunological importance.
Group 3 innate lymphoid cells produce the growth factor HB-EGF to protect the intestine from TNF-mediated inflammation.
Innate Lymphocyte/Ly6C(hi) Monocyte Crosstalk Promotes Klebsiella Pneumoniae Clearance.
The Role of IgG Fc Region N-Glycosylation in the Pathomechanism of Rheumatoid Arthritis.
Tumor Necrosis Factor Alpha-Induced Interleukin-1 Alpha Synthesis and Cell Death Is Increased in Mouse Epithelial Cells Infected With Chlamydia muridarum.
Customizable Multiplex Antibody Array Immunoassays with Attomolar Sensitivities.
[Tumor necrosis factor alpha (TNF-α) in autoimmune diseases (AIDs): molecular biology and genetics].
The role of interleukins and their inhibitors in the development of autoimmune uveitis.
Tumor necrosis factor alpha in mycobacterial infection.
Functional characterization of a putative lipopolysaccharide-induced TNF-alpha factor (LITAF) from blood clam Tegillarca granosa in innate immunity.