Spontaneous changes in ventricular tachycardia cycle length and their relation to earliest sites of epicardial activation in a canine model

Gómez, María de Jesús; Helie, Francois; Sierra, Gilberto; Rocque, Pierre; Vinet, Alain; Cardinal, René; Nadeau, Réginald.
Arch. Inst. Cardiol. Méx ; 70(1): 9-18, ene.-feb. 2000. ilus, graf
Artículo en Inglés | LILACS | ID: lil-280387

Documentos relacionados

  1. Fat deposition and repolarization: another brick in the wall promoting ventricular tachycardia?
  2. Polymorphic Ventricular Tachycardia: Terminology, Mechanism, Diagnosis, and Emergency Therapy.
  3. Experimental verification of the value of the T<sub>peak</sub> -T<sub>end</sub> interval in ventricular arrhythmia inducibility in an early repolarization syndrome model.
  4. Increased Local Sympathetic Nerve Activity During Pathogenesis of Ventricular Arrhythmias Originating from the Right Ventricular Outflow Tract.
  5. ß<sub>1</sub> -Adrenoceptor autoantibodies increase the susceptibility to ventricular arrhythmias involving abnormal repolarization in guinea-pigs.
  6. Idiopathic epicardial ventricular tachycardia from the coronary venous system: From electrocardiographic recognition to appropriate therapy.
  7. High-density mapping revealing the complex nature of a figure-of-eight re-entrant ventricular tachycardia with endocardial isthmus and double epicardial exit.
  8. Masked hypertension is related to alteration of myocardial arrhythmia Parameters.
  9. Cardiac sympathetic denervation: A wonder solution to channelopathy-related ventricular tachycardia and much more.
  10. IL-18 mediates sickle cell cardiomyopathy and ventricular arrhythmias.