Abnormal subcellular distribution of GLUT4 protein in obese and insulin-treated diabetic female dogs
Vargas, A. M; Barros, R. P. A; Zampieri, R. A; Okamoto, M. M; De Carvalho Papa, P; Machado, U. F.
Braz. j. med. biol. res
; 37(7): 1095-1101, July 2004. ilus, tab, graf
Artículo en Inglés | LILACS | ID: lil-360936
Documentos relacionados
Knockout of syntaxin-4 in 3T3-L1 adipocytes reveals new insight into GLUT4 trafficking and adiponectin secretion.
The people behind the papers - Gabriela Krejcová and Adam Bajgar.
Probing Insulin Sensitivity with Metabolically Competent Human Stem Cell-Derived White Adipose Tissue Microphysiological Systems.
Insulin-stimulated adipocytes secrete lactate to promote endothelial fatty acid uptake and transport.
Phospholipase C-related but catalytically inactive protein acts as a positive regulator of insulin signalling in adipocytes.
Analytical comparison of absolute quantification strategies to investigate the insulin signaling pathway in fat cells.
Bisphenol F suppresses insulin-stimulated glucose metabolism in adipocytes by inhibiting IRS-1/PI3K/AKT pathway.
Characterization of lipoprotein lipase storage vesicles in 3T3-L1 adipocytes.
GALNT2 as a novel modulator of adipogenesis and adipocyte insulin signaling.
<i>FTO</i> promoted adipocyte differentiation by regulating <i>ADRB1</i> gene through m<sup>6</sup>A modification in Hycole rabbits.