In situ cellular immune response in non-ulcerated skin lesions due to Leishmania (L) infantum chagasi infection

Sandoval, Carmen; Araujo, Gabriela; Sosa, Wilfredo; Avalos, Sara; Silveira, Fernando; Corbett, Carlos; Zúniga, Concepción; Laurenti, Marcia.
J. Venom. Anim. Toxins incl. Trop. Dis. ; 27: e20200149, 2021. graf, ilus
Artículo en Inglés | VETINDEX | ID: vti-31984

Documentos relacionados

  1. In situ cellular immune response in non-ulcerated skin lesions due to Leishmania (L) infantum chagasi infection
  2. Paradoxical immune response in leishmaniasis: The role of toll-like receptors in disease progression.
  3. Immune-metabolic interactions between Leishmania and macrophage host.
  4. Aberrant expression of SOCS impairs the anti-leishmanial immune response.
  5. Skin regeneration of amphibians: A novel model for skin regeneration as adults.
  6. Transcription Factors Interplay Orchestrates the Immune-Metabolic Response of <i>Leishmania</i> Infected Macrophages.
  7. Leishmania amazonensis infection regulates oxidate stress in hyperglycemia and diabetes impairing macrophage's function and immune response.
  8. Understanding the immune responses involved in mediating protection or immunopathology during leishmaniasis.
  9. Protective effects of dissolved molecular hydrogen against hydrogen peroxide-, hydroperoxide-, and glyoxal-induced injuries to human skin keratinocytes.
  10. Hypoxia inducible factor 1&#945;: A critical factor for the immune response to pathogens and Leishmania.