Relationship between P65 and radiotherapy-induced oral squamous cell carcinoma cell line apoptosis
Li MA; Dong-sheng ZHANG; Jun-lou WU; Jun-qing HAN; Shi-zhou ZHANG; Gui-jun LIU; Wen-li MU; Jie ZHANG.
West China Journal of Stomatology
; (6): 318-322, 2011.
Artículo en Zh | WPRIM | ID: wpr-235056
Documentos relacionados
[Relationship between P65 and radiotherapy-induced oral squamous cell carcinoma cell line apoptosis].
NF-κB subunits direct kinetically distinct transcriptional cascades in antigen receptor-activated B cells.
RELA promotes the progression of oral squamous cell carcinoma via TFAP2A-Wnt/ß-catenin signaling.
RELA governs a network of islet-specific metabolic genes necessary for beta cell function.
NFκB (RelA) mediates transactivation of hnRNPD in oral cancer cells.
ZFTA::RELA fusion in a distinct liposarcoma morphologically overlapping with chondroid lipoma.
Enhanced transcriptional heterogeneity mediated by NF-κB super-enhancers.
Enhancement of a prime editing system via optimal recruitment of the pioneer transcription factor P65.
The Small Molecule BC-2059 Inhibits Wingless/Integrated (Wnt)-Dependent Gene Transcription in Cancer through Disruption of the Transducin <i>ß</i>-Like 1-<i>ß</i>-Catenin Protein Complex.
RvD1<sub>n-3 DPA</sub> Downregulates the Transcription of Pro-Inflammatory Genes in Oral Epithelial Cells and Reverses Nuclear Translocation of Transcription Factor p65 after TNF-α Stimulation.