Human papillomavirus type 16 E5 sequence evolution analysis
Min HAO; Xiao-zhou WU; Xiao-li WANG; Mei-lu BIAN; Hong-tu LIU.
Chinese Journal of Experimental and Clinical Virology
; (6): 452-454, 2009.
ArtÃculo en Zh | WPRIM | ID: wpr-325514
Documentos relacionados
Crowd Control: E7 Conservation Is the Key to Cancer.
Longitudinal follow-up of HPV16 sequence after cervical infection: Low intrahost variation and no correlation with clinical evolution.
Serologic Response to Human Papillomavirus Genotypes Among Unvaccinated Women: Findings From the HITCH Cohort Study.
[Early Mitotic Inhibitor 1 Regulates DNA Re-replication Mediated by Human Papillomavirus Subtype 16 E7 in Response to DNA Damage].
Two conserved amino acids differentiate the biology of high-risk and low-risk HPV E5 proteins.
Evidence for direct interaction between the oncogenic proteins E6 and E7 of high-risk human papillomavirus (HPV).
A novel HPV16 splicing enhancer critical for viral oncogene expression and cell immortalization.
Advances in molecular mechanism of HPV16 E5 oncoprotein carcinogenesis.
The polymorphism analysis and epitope predicted of Alphapapillomavirus 9 E6 in Sichuan, China.
In silico prediction of T-cell and B-cell epitopes of human papillomavirus type 16 L1 protein.