An Analysis of Movement Function of SD Rats'Spinal Cord Injury After Joint Therapy Using NT- 3- HUMSCs and SOCS3 Gene Silencing
Gang BAI; Hong-Tian ZHANG; Jun LAI; Lin LUO; Pin ZUO; Yao-Dong FAN.
Journal of Kunming Medical University
; (12): 15-20, 2018.
ArtÃculo en Zh | WPRIM | ID: wpr-694523
Documentos relacionados
Joint Therapy by NT-3-HUMSCs and SOCS3 Gene Dilencing in Nerve Regeneration Repair after Spinal Cord Injury in SD Rats
AAV Vector Mediated Delivery of NG2 Function Neutralizing Antibody and Neurotrophin NT-3 Improves Synaptic Transmission, Locomotion, and Urinary Tract Function after Spinal Cord Contusion Injury in Adult Rats.
Repair effect of neurotrophic factor III (NT-3) on rats with spinal injury model and its mechanism.
Transplantation of neurotrophin-3-expressing bone mesenchymal stem cells improves recovery in a rat model of spinal cord injury.
[Treatment of autotransplantation of NT-3 gene modified OECs and NSCs complex adhering to scaffold on spontaneous cerebral hemorrhage].
Effect of NT-3 on repair of spinal cord injury through the MAPK signaling pathway.
Exogenous Expression of Nt-3 and TrkC Genes in Bone Marrow Stromal Cells Elevated the Survival Rate of the Cells in the Course of Neural Differentiation.
Neurotrophin-3 promotes peripheral nerve regeneration by maintaining a repair state of Schwann cells after chronic denervation via the TrkC/ERK/c-Jun pathway.
[Protective effects of pretreatment with neurotrophin-3 on intrathecal ropivacaine in rats].