ABSTRACT
This study presents a case report of the clinical presentation and management of a 47-year-old male myope who underwent photorefractive keratectomy (PRK) and developed bilateral sterile corneal infiltrates at 1 day post-operatively. The patient was successfully treated with aggressive topical antibiotic and topical steroid therapy. The final corrected distance visual acuity (CDVA) was 20/25 with faint corneal scarring. Peripheral sterile corneal infiltrate can occur after PRK with excellent prognosis. Infectious causes should be suspected in all cases of corneal infiltrate. The most likely cause of peripheral sterile corneal infiltrate in this case was pooling of the tear film containing antigens under the bandage contact lens.
Subject(s)
Keratitis/etiology , Photorefractive Keratectomy/adverse effects , Anti-Bacterial Agents/therapeutic use , Astigmatism/surgery , Drug Therapy, Combination , Glucocorticoids/administration & dosage , Humans , Keratitis/drug therapy , Lasers, Excimer/therapeutic use , Male , Middle Aged , Myopia/surgery , Ofloxacin/administration & dosage , Visual Acuity/physiologyABSTRACT
PURPOSE: To report 6 eyes of 5 patients with transient corneal edema after exposure to the milky latex of Calotropis procera (ushaar). METHODS: Interventional case series. RESULTS: Intracorneal penetration of ushaar latex can lead to permanent endothelial cell loss with morphologic alteration. Corneal edema resolved completely in approximately 2 weeks in all cases, despite reduced endothelial cell count and abnormal morphology. CONCLUSIONS: Corneal endothelial toxicity of ushaar latex is caused by its ability to penetrate the corneal stroma and induce permanent loss of endothelial cells. Corneal edema resolves if sufficient endothelial cell viability is still present after resolution of ushaar keratitis.
