Corrigendum: The relationship between locus of control and pre-competitive anxiety in highly trained soccer players.

[This corrects the article DOI: 10.3389/fpsyg.2023.1227571.].

The relationship between locus of control and pre-competitive anxiety in highly trained soccer players.

Introduction: Previous studies have not considered the potential association between locus of control and precompetitive anxiety in elite soccer players. Accordingly, this cross-sectional study examined The prediction of locus of control on precompetitive anxiety in highly trained cadet soccer players. Objective: Based on a literature review, our research question was: can the locus of control be considered as an explanatory element of precompetitive anxiety? Methods: Thirty-five Tunisian highly trained soccer players licensed from two regional soccer clubs aged between 15 and 16 years participated in the resent study. All participants were evaluated using the Competitive State Anxiety Inventory-2 (CSAI-2) and the Internal-External Locus of Control Scale. The relationship between measures of anxiety, self-confidence and the locus of control scores were analyzed using Pearson's product-moment correlation coefficient. Further, multiple linear stepwise multiple regression models were calculated to determine the most robust predictors of the locus of control. Results: Based on our findings, the regression analysis explains up to 21.3% of the total variation of our independent variable (locus of control) and explains only 21.3% of the variability of our dependent variable somatic anxiety. Furthermore, locus of control explains 61.9% of the variability in self-confidence. Conclusion: The locus of control can be used for the detection and selection of young athletic talent to identify individuals with the best psychological aptitude to cope with psychological problems related to sports performance. Preparing highly trained soccer players on how to deal with their anxiety could prevent them from becoming overwhelmed when they feel powerless to change their situation during competition.

High frequency of the p.R34X mutation in the TMC1 gene associated with nonsyndromic hearing loss is due to founder effects.

Founder mutations, particularly 35delG in the GJB2 gene, have to a large extent contributed to the high frequency of autosomal recessive nonsyndromic hearing loss (ARNSHL). Mutations in transmembrane channel-like gene 1 (TMC1) cause ARNSHL. The p.R34X mutation is the most frequent known mutation in the TMC1 gene. To study the origin of this mutation and determine whether it arose in a common ancestor, we analyzed 21 polymorphic markers spanning the TMC1 gene in 11 unrelated individuals from Algeria, Iran, Iraq, Lebanon, Pakistan, Tunisia, and Turkey who carry this mutation. In nine individuals, we observed significant linkage disequilibrium between p.R34X and five polymorphic markers within a 220 kb interval, suggesting that p.R34X arose from a common founder. We estimated the age of this mutation to be between 1075 and 1900 years, perhaps spreading along the third Hadramaout population movements during the seventh century. A second founder effect was observed in Turkish and Lebanese individuals with markers in a 920 kb interval. Screening for the TMC1 p.R34X mutation is indicated in the genetic evaluation of persons with ARNSHL from North African and Southwest Asia.