1.
Bull Exp Biol Med
; 165(1): 48-51, 2018 May.
Article
in English
| MEDLINE
| ID: mdl-29796808
ABSTRACT
Light microscopy, immunohistochemistry, and morphometric examinations established that cell death in lung granulomas of BCG-infected mice resulted mainly from activation of receptor-mediated apoptosis, which did not prevent the persistence of the causative agent in macrophages of the granulomas and promoted the formation of pronounced fibrosis in granulomas and pulmonary interstitium.