ABSTRACT
Este estudo teve como objetivo interpretar e analisar como o Jornal Folha de São Paulo retratou os Jogos Olímpicos Rio 2016 como evento esportivo na cobertura esportivo-midiática da Folha de São Paulo. Este artigo é resultado de uma pesquisa qualitativa descritiva, tendo como corpus o Jornal Folha de São Paulo, referente às edições de 1º de julho a 30 de setembro de 2016. A análise deu-se através de períodos temporais, de acordo com o que estava acontecendo em cada período. A cobertura do pré-evento muito se preocupou com o término das estruturas e se o país tinha condições de sediar JO. Por outro lado, a cobertura durante o evento mostrou que o país tinha condições de sediar megaeventos. Por fim, a cobertura do pós-evento retratou o legado que os jogos deixaram para o país sede das OlimpíadasRio-2016.
This study aimed to interpret and analyze how the newspaper Folha de São Pauloportrayed Rio 2016 Olympic Games as a sports event in the sports media coverage of Folha de São Paulo. This paper is the result of a qualitative descriptive research, using as corpusthe newspaper Folha de São Paulo, editions from July 1stto September 30thof 2016. The analysis was carried out through time periods, according towhat was happening in each period. The pre-event coverage was really concerned about the structure completion and whether the country was ready to host the OG. On the other hand, the coverage during the event showed that the country had the ability to host mega-events. Lastly, the post-event coverage portrayed the legacy that the games left to the host country of Rio 2016 Olympics.
Subject(s)
Play and Playthings , SportsABSTRACT
Studies indicate that histone deacetylation is important for long term changes related to stress and antidepressant treatment. The present study aimed to evaluate the effects of the classic antidepressant imipramine, and of an antagonist of the N-methyl-d-asparte (NMDA) receptor, ketamine, on behavior and histone deacetylase (HDAC) activity in the brains of maternally deprived adult rats. To this aim, deprived and non-deprived (control) male Wistar rats were divided into the following groups: non-deprived+saline; non-deprived+imipramine (30 mg/kg); non-deprived+ketamine (15 mg/kg); deprived+saline; deprived+imipramine (30 mg/kg); and deprived+ketamine (15 mg/kg). The drugs were administrated once a day for 14 days during their adult phase. Their behavior were then assessed using the forced swimming and open field tests. In addition, the HDAC activity was evaluated in the prefrontal cortex, hippocampus, amygdala and nucleus accumbens using the kit ELISA-sandwich test. In deprived rats treated with saline, we observed an increase in the immobility time, but treatments with imipramine and ketamine were able to reverse this alteration, decreasing the immobility time. Also, there was a decrease on number of crossings with imipramine treatment in non-deprived rats, and an increase on number of crossings with ketamine treatment in deprived rats. The HDAC activity did not alter in the prefrontal cortex, hippocampus and amygdala by deprivation or via treatment with imipramine or ketamine. However, in the nucleus accumbens we observed an increase of HDAC activity in the deprived rats, and interestingly, imipramine and ketamine treatments were able to decrease HDAC activity in this brain area. These findings provide a novel insight into the epigenetic regulation of histone deacetylase in the nucleus accumbens caused by imipramine and ketamine, and indicate that molecular events are necessary to reverse specific stress-induced behavior.
