Applied load on the horse's back under racing conditions.

With the intention of limiting the weight on horses' backs and guaranteeing maximal freedom of movement, commonly used racing saddles are small and have minimal cushioning. Poor saddle cushioning may limit performance or even affect soundness of the back. The aim of this study was to measure the pressure under an average racing saddle ridden by a jockey at racing speed. Saddle pressure using a medium-sized racing saddle (length 37 cm, weight 450 g) was measured in five actively racing Thoroughbred horses. All horses were trained at the same facility and ridden by their usual professional jockey, weighing 60 kg. The horses were ridden on a race track at canter (mean velocity, V1 ± standard deviation, SD: 7.7 ± 0.4m/s) and gallop (V2 ± SD: 14.0 ± 0.7 m/s). Maximal pressure was 134 kPa at V1 and 116 kPa at V2. Mean peak pressure was 73.6 kPa at V1 and 54.8 kPa at V2. The maximal total force did not differ between the two velocities and was approximately twice the jockey's bodyweight. The centre of pressure lateral range of motion differed significantly, with excursions of 23 mm at V1 and 37 mm at V2; longitudinal excursion was 13 mm for V1 and 14 mm for V2. The highest pressure (>35 kPa) was always localised along the spinous processes over an average length of 12.5 cm. It was concluded that racing saddles exert high peak pressures over bony prominences known to be sensitive to pressure.

Atrial natriuretic peptide and cardiac troponin I concentrations in healthy Warmblood horses and in Warmblood horses with mitral regurgitation at rest and after exercise.

OBJECTIVE: Atrial natriuretic peptide (ANP) and cardiac troponin I (cTnI) serve as biomarkers for increased cardiac pressure/volume loading and for myocardial stress or damage. The objective was to describe the time course of plasma ANP concentrations (CpANP) and plasma cTnI concentrations (CpcTnI) in horses with mitral regurgitation (MR) compared to healthy horses at rest and after exercise, and to describe the relationship of CpANP with cardiac dimensions and intracardiac pressures. ANIMALS: 15 healthy Warmblood horses and 7 Warmblood horses with MR. METHODS: Cardiac dimensions at rest were measured using echocardiography. All horses underwent standardized treadmill exercise. Biomarker concentrations and intracardiac pressures were measured at rest and after exercise. Hypotheses were tested using statistical methods. The level of significance was P < 0.05. RESULTS: Horses with MR showed increased left atrial (LA) and left ventricular (LV) dimensions but similar exercise capacity compared to healthy horses. Pulmonary capillary wedge pressures (PCWP) and CpANP increased with exercise. Horses with MR had higher PCWP and higher CpANP at rest and after exercise compared to healthy horses, with the maximum difference in CpANP reached 10 min after exercise. CpANP was significantly related to PCWP and - although inconsistently and only in healthy horses - to echocardiographic indices of LA and LV size and function. CpcTnI was low throughout the study in both groups. CONCLUSIONS: CpANP is increased in horses with MR and is related to LA pressures and to left heart dimensions. MR is not necessarily associated with exercise intolerance and exercise-induced myocardial stress or damage.