ABSTRACT
INTRODUCTION: Eighty-five per cent of uterine inversions are puerperal. Non-puerperal uterine inversion is usually caused by tumours that exert a traction force on the fundus of the uterus. This causes the uterus to be partially or completely inverted. It is commonly related to benign tumours like submucosal leiomyomas. Nevertheless, malignancies are an infrequent association. CASE PRESENTATION: We report a case of a 35-year-old female patient, medically and surgically free, gravida0 para0, complaining of menometrorrhagia associated with pelvic pain for 2 years. A suprapubic ultrasound scan showed an enlarged, globular uterus with a heterogeneous, undefined mass of 49 mm in size. MRI scan showed the appearance of a U-shaped uterine cavity and a thickened inverted uterine fundus with an endometrial infiltrating mass of 25 mm. Intraoperative exploration showed uterine inversion involving the ovaries; the fallopian tubes and the round ligaments and a necrotic intracavitary mass. The malignancy of the tumor was confirmed through anatomopathological examination as Adenosarcoma. CONCLUSIONS: Uterine inversion is rare outside the puerperal period, and malignant etiology must not be overlooked. Therefore, comprehensive care with meticulous etiological investigation is crucial.
Subject(s)
Adenosarcoma , Leiomyoma , Urogenital Abnormalities , Uterine Inversion , Uterine Neoplasms , Uterus/abnormalities , Female , Humans , Adult , Uterine Inversion/diagnosis , Uterine Inversion/etiology , Uterine Inversion/surgery , Uterine Neoplasms/complications , Uterine Neoplasms/diagnosis , Uterine Neoplasms/surgery , Adenosarcoma/complications , Adenosarcoma/diagnosis , Adenosarcoma/surgery , Leiomyoma/surgeryABSTRACT
INTRODUCTION AND IMPORTANCE: Hydatid cysts are often found in the liver or lungs. As for the parotid localization, it remains exceptional even in endemic areas and presents as a tumor of the parotid thus representing a challenge to the practitioner. CASE PRESENTATION: A 54-year-old male farmer presented with a swelling in the left parotid gland region evolving over the past 2 years. Physical examination revealed a non-tender and mobile swelling in the left parotid region, the overlying skin was normal. Magnetic resonance imaging showed a cystic lesion of the left parotid gland. Fine-needle aspiration cytology (FNAC) yielded a clear fluid with a few benign epithelial cell. A superficial parotidectomy was performed. Histopathological examination confirmed the presence of a hydatid cyst in the parotid gland. CLINICAL DISCUSSION: Parotid hydatid cyst is exceptional. However, it can be suspected in the context of an isolated parotid cystic mass in an endemic area, even in the absence of any other associated location. CONCLUSION: Hydatid cysts of the parotid gland have a polymorphic and a specific symptomatology and require surgical removal.
ABSTRACT
INTRODUCTION AND IMPORTANCE: Superior ophthalmic vein thrombosis (SOVT) is a very rare condition that can occur following either septic or aspetic causes. Early recognition and management is important in order to avoid vision-threatening and fatal complications. CASE PRESENTATION: A 28-year-old man presented with orbital cellulitis complicating acute pansinusitis. CT scan revealed ipsilateral SOVT. The patient received intravenous antibiotics and anticoagulation with strict ophthalmic monitoring. No orbital complications were recorded during the course of treatment. CLINICAL DISCUSSION: SOVT is caused either by septic or aseptic causes. Treatment depends on the etiology. Corticosteroids and anticoagulants are controversial. CONCLUSION: Early diagnosis is key to successful management of SOVT.
ABSTRACT
Inner ear hemorrhage is an extremely rare cause of sudden sensorineural hearing loss with few cases reported in the literature. We report the case of a 30-year-old male who presented with a sudden left ear hearing loss, with no tinnitus nor vertigo. The audiogram revealed a profound left sensorineural hearing loss. An MRI of the brain and internal auditory canal was performed 3 weeks after and revealed an increased signal intensity on T1-weighted (T1W) and T2 fluid-attenuated inversion recovery (FLAIR) images in the left cochlea. No other abnormalities were found, in particular no enhancement after intravenous administration of gadolinium. The CISS 3D sequence showed a signal of discreetly lower intensity in the left cochlea compared to the right one. The diagnosis of intracochlear hemorrhage was made. No improvement of the hearing loss has been noted after medical treatment and hyperbaric oxygen therapy.
