ABSTRACT
By means of histological and histochemical methods slices of biopsies of the canine musculus latissimus dorsi have been investigated after electroneurostimulation for three months through the thoracodorsal nerve in situ and after cutting its initial part. Frequency of contractions increases gradually from 30 up to 80 per 1 min every 2 weeks. The preparations are stained with hematoxylin--eosin. Histochemical reactions for adenosinetriphosphatase (ATPase) (incubational medium pH 4.3 and 10.3) and for acetylcholinesterase (AChE) are performed. The muscle, stimulated in situ, preserves its normal structure. Transformation of muscle contractions from fast to slow, as well as increasing AChE activity in myoneural synapses are revealed. The muscle, stimulated after its cutting at its beginning, is subjected to adipose dystrophy. Activity of ATPase and AChE do not differ from that in the control.
Subject(s)
Muscles/physiopathology , Transcutaneous Electric Nerve Stimulation/methods , Acetylcholinesterase/metabolism , Adenosine Triphosphatases/metabolism , Animals , Biopsy , Dogs , Electrodes, Implanted , Histocytochemistry , Muscles/enzymology , Neuromuscular Junction/enzymology , Time FactorsSubject(s)
Aluminum Oxide/adverse effects , Aluminum/adverse effects , Bone and Bones/drug effects , Ceramics/adverse effects , Dental Implantation, Endosseous/instrumentation , Dental Materials/toxicity , Animals , Equipment Design , Femur/drug effects , Mandible/drug effects , Materials Testing , Rabbits , Time FactorsABSTRACT
By means of postmortem selective coronary angiography coronary collaterals of 55 sudden out-of-hospital coronary death victims were investigated. The collaterals were found in most (58,5%) sudden coronary death cases. There was no correlation between the presence of coronary collaterals, on the one hand, and ischaemic heart disease symptoms during the life and focal myocardial lesions in deceased persons, on the other. The investigation showed the maximal formation of collaterals in cases with asymmetrical obstructive lesions of the right and left coronary arteries. This is in agreement with a hypothesis claiming the coronary collateral formation to be dependent on the pressure gradient between individual coronary arteries.
