ABSTRACT
A new spin- and angle-resolved inverse photoemission setup with a low-energy electron source is presented. The spin-polarized electron source, with a compact design, can decouple the spin polarization vector from the electron beam propagation vector, allowing one to explore any spin orientation at any wavevector in angle-resolved inverse photoemission. The beam polarization can be tuned to any preferred direction with a shielded electron optical system, preserving the parallel beam condition. We demonstrate the performances of the setup by measurements on Cu(001) and Au(111). We estimate the energy resolution of the overall system at room temperature to be â¼170 meV from kBTeff of a Cu(001) Fermi level, allowing a direct comparison to photoemission. The spin-resolved operation of the setup has been demonstrated by measuring the Rashba splitting of the Au(111) Shockley surface state. The effective polarization of the electron beam is P = 30% ± 3%, and the wavevector resolution is ΔkF â² 0.06 Å-1. Measurements on the Au(111) surface state demonstrate how the electron beam polarization direction can be tuned in the three spatial dimensions. The maximum of the spin asymmetry is reached when the electron beam polarization is aligned with the in-plane spin polarization of the Au(111) surface state.
ABSTRACT
In contrast with senescence-accelerated mice R1, SAM P8 show abnormal aging characteristics. Changes occurring during aging could be mainly caused by free radical reactions. The brain is a plasmalogen-rich tissue. These particular phospholipids may act as endogenous antioxidants, be oxidized and release long chain aldehydes and alpha-hydroxyaldehydes during oxidative stress. The aim of this study was to examine by GC/MS the age- and strain-related levels of plasmalogens, aldehydes and alpha-hydroxyaldehydes in brain homogenates of SAM P8 and R1 at weaning, 5 months and 9 months of age in order to better understand the differences between both strains. In SAM R1, the evolution of brain plasmalogen levels corresponded to characteristics of normal aging: an increase from weaned to adult mice followed by a decrease characterizing the normal loss of myelin. By contrast to SAM R1, there was no change in the plasmalogen content in SAM P8 brain. The levels of aldehydes and alpha-hydroxyaldehydes were similar for both strains, they remained constant between adult and aged mice. Specific changes in the aging of SAM P8 were not explained by cerebral levels of these oxidative products. Other mechanisms related to the toxicity of aldehydes and alpha-hydroxyaldehydes could be considered.
