ABSTRACT
OBJECTIVE: The aim of this study was to assess the frequency of cement residues after cementation of CAD/CAM monolithic zirconia crowns on customized CAD/CAM titanium abutments. MATERIALS AND METHODS: Sixty premolars and molars were restored on Astra Tech Osseospeed TX™ implants using single monolithic zirconia crowns fixed on two types of custom-made abutments: Atlantis™ titanium or Atlantis™ Gold Hue. Occlusal openings providing access to the abutment screws were designed for retrievability of the crown/abutment connection. After fixation with glass ionomer cement, the crown/abutment units were unscrewed to evaluate the presence of residual cement. Dichotomous assessment of the presence or absence of cement at the crown/abutment unit and peri-implant tissues was performed. RESULTS: Clinically undetected cement excess was visible on 44 of 60 restorations (73.3%). There was no interdependency between residual cement presence and implant location or diameter. However, a dependency between the presence of residual cement and the aspect of the abutment/crown connection could be noted. The majority of the residues were observed on the distal (17.9%) and mesial (15%) aspects. While on the palatal/lingual aspect, the cement was visible in 8.8%; only 3.4% of all surfaces displayed cement residues. CONCLUSIONS: Within the limitations of the study, it can be concluded that the use of customized CAD/CAM abutments do not guarantee avoidance of subgingival cement residues after crown cementation.
Subject(s)
Computer-Aided Design , Crowns , Dental Abutments , Dental Cements , Dental Prosthesis Design , Dental Prosthesis Retention/methods , Bicuspid , Female , Glass Ionomer Cements/chemistry , Humans , Male , Middle Aged , Molar , Prospective Studies , Titanium , ZirconiumABSTRACT
Cieszynrski was a Polish dentist who formulated the rules of isometry (Cieszyrnski's isometry) in dental radiology, which enables precise dental X-ray pictures that show the true dimensions of teeth. Cieszynski also developed an extra-oral technique of anesthetizing the mandibular alveolar nerve, and a treatment for trigeminal neuralgia which involved injections of alcohol to the Gasser's ganglion. The clinical activity of Antoni Cieszynski included periodontal surgery (Cieszynski-Widmann-Neumann's operation) and surgery of abscesses and phlegmones of head and neck. He was also a member of numerous scientific societies (ADA, ASI, FDI) and a founder of the first Polish dental journal Polska Dentystyka (Polish Dentistry). On the 70th anniversary of Doctor Antoni Cieszynski's tragic death, the authors recall his outstanding and fruitful clinical career.
Subject(s)
Anesthesia, Dental/methods , Anesthesiology/history , Periodontics/history , Radiography, Dental/methods , Radiology/history , Surgery, Oral/history , History, 19th Century , History, 20th Century , Poland , Radiotherapy/history , World War IIABSTRACT
OBJECTIVES: This review groups the newest results of molecular analyses of DSPP gene for patients diagnosed either with dentinogenesis imperfecta type II/III or dentine dysplasia and tries to link the phenotypes with specific mutations in the DSPP gene. DATA: The review includes biochemical data introducing a specificity of DSPP protein which justifies it as a critical factor for dentine mineralization and maturation. The majority of the review analyzes mutations in the DSPP gene which result in phenotypes of dentinogenesis imperfecta types II or/and III or dentine dysplasia. SOURCES: An electronic search was conducted in the databases of Pub Med and supplemented by manual study of relevant references. STUDY SELECTION: 52 out of 108 references were finally selected for the review based on the novelty and/or originality of data. CONCLUSION: Hereditary dentine disorders dentinogenesis imperfecta type II/III and dentine dysplasia are currently proposed to be one disease with distinct clinical manifestations reflecting various mutations in the same DSPP gene. For years both disorders were linked exclusively to mutations in the DSP code but a growing number of papers describe mutations which manifest a similar phenotype but are localized in the strongly repetitive sequence of the 3' terminus of the DSPP which codes DPP protein. Our search suggests that the localization of mutation in the sequence of the DSPP gene might result in a different phenotype due to the diverse cellular fate of the mutated protein. Thus comprehensive research on the cellular fate and processing of both normal and mutated DSPP is still required.
