Optical, Electrical, and Morphologic Characterization of Chromium-Impurified CdS Thin Films Elaborated by the Chemical Bath Deposition.

In this research work, a material system formed of cadmium sulfide combined with chromium atoms was developed to evaluate the influence of chromium concentration on the optical, electrical, structural, and morphological properties of a precursor layer of CdS. It is possible to observe that the transmission spectra increased for all chromium concentrations analyzed. From X-ray diffractograms, we conclude more accurately that CdS presents a mixture of phases, including orthorhombic, hexagonal, and cubic. Furthermore, the impact of adding chromium results in variations in the intensity of two major peaks in the diffractograms and an anomalous shift in the CdS pattern. The calculated resistivities show an invariable behavior of 4.5 × 106 Ω cm. In addition, the bandgap values remain practically constant, with values of approximately 2.43-2.44 eV. The addition of chromium at different concentrations leads to surface morphology changes, as observed in SEM images.

[Calcified aortic stenosis due to healed experimental bacterial endocarditis]. / Estenosis aórtica calcificada por endocarditis bacteriana experimental curada.

We studied the role of bacterial endocarditis in the development of aortic valve stenosis. A femoral arterio venous shunt was performed in nine dogs with the method previously proposed by Lillehei. We induced bacteremic infection with the administration of streptococcus mitis (1 x 10(10)) 10 ml once a day for 15 days these bacterium were sensible to penicillin. All dogs were treated with 1,000,000 U of benzatinic penicillin and sacrificed between 28-102 days after the bacterial inoculation ended. In one dog we observed bacterial endocarditis in the mitral and aortic valves and in other three dogs there was an aortic valve stenosis with calcium deposits in the body and in the free edges of the aortic valve with evident irregular stenosis as seen in man.

[Uterine leiomyosarcoma which spread through the vena cava to the pulmonary artery]. / Leiomiosarcoma uterino propagado por vena cava hasta arteria pulmonar.

We describe a case of uterine leiomyosarcoma with four elongations that, through the vena cava, reached the pulmonary artery, left atrium, and right renal artery respectively. Clinical features suggested congenital pulmonary artery stenosis with pulmonary emboli. Clinical diagnosis was not made in spite of angiography and radionuclide scans. We review the literature on the subject. We also discuss the amenability of surgical correction as well as the mechanisms of the intravenous growth of this type of tumor.

[Primary mesothelioma of the pericardium. Report of 2 cases and review of the literature]. / Mesotelioma primario del pericardio. Comunicación de dos casos y revisión de la literatura.

We described two adult females with primary pericardial mesothelioma. There was not history of asbestos exposure. The clinical picture suggested pericardial constriction, with predominantly right sided heart failure. Anatomically; one case was of a nodular type, being the other diffuse. On histology both cases were of the fibrous type. The second case was less differentiated; it perforated the right atrial wall creating an intracavitary mass. A review of the literature is made.

[Pathology of the papillary muscles]. / Patología de los músculos papilares.

Anatomoclinical findings in 21 cases of papillary muscle (PM) damage found in 4,000 consecutive autopsies are reviewed. Mitral PM muscles were involved in 17 cases, the posteromedial muscle in 11 patients, in 8 of these with necrosis (N) in 3 with fibrosis (F), in 2 with rupture. The mitral antero-lateral (MAL) was involved in 4 cases, by myocardial infarction (MI), fibrosis, hemorrhage and infectious endocarditis (IE). Both mitral PM were involved in two patients (MI and endomyocardial fibrosis). Tricuspid PM involvement was found in 4 cases. In two because of MI and the two remaining with fibrosis. In the 12 cases with MI microscopic examination showed, PM necrosis of variable magnitude: in the 7 cases with F, there was increased interstitial tissue by connective tissue; in the case with IE there was severe inflammatory reaction with microabscess formation and in one case, a small central longitudinal artery was damaged, a finding not previously described. Mitral PM infarction caused regurgitant murmurs, increase in heart size, heart failure and death from acute pulmonary edema or serious arrhythmias. There was no radiological evidence of left atrial enlargement. ECG findings were not useful in the diagnosis. Based on this anatomoclinical experience considerations are made about other diagnostic and surgical methods.