Subject(s)
Breath Tests/instrumentation , Nitric Oxide/metabolism , Adolescent , Adult , Aged , Asthma/diagnosis , Asthma/metabolism , Eosinophilia/diagnosis , Eosinophilia/metabolism , Exhalation , Female , Humans , Male , Middle Aged , Reproducibility of Results , Young AdultABSTRACT
No disponible
Subject(s)
Humans , Male , Female , Adolescent , Young Adult , Adult , Middle Aged , Aged , Nitric Oxide/blood , Breath Tests/instrumentation , Asthma/blood , Reproducibility of Results , Cross-Sectional Studies , Biomarkers/blood , Reference StandardsSubject(s)
Anti-Asthmatic Agents/therapeutic use , Asthma/drug therapy , Asthma/etiology , Eosinophils/immunology , Anti-Asthmatic Agents/pharmacology , Asthma/diagnosis , Biological Therapy/methods , Biomarkers , Eosinophils/metabolism , Eosinophils/pathology , Female , Humans , Leukocyte Count , Male , Middle Aged , Treatment OutcomeABSTRACT
No disponible
Subject(s)
Humans , Male , Female , Middle Aged , Asthma/drug therapy , Asthma/immunology , Biological Therapy/methods , Severity of Illness Index , Treatment OutcomeABSTRACT
Idiopathic pylephlebitis and primary sclerosing peritonitis are two highly unusual entities. To our knowledge, the association of the two diseases has not been described previously. We report a 42-year-old patient with a protein S deficiency who presented with fever and chills, in whom idiopathic pylephlebitis was diagnosed. A year later, the patient was readmitted because of recurrent vomiting and weight loss. An exploratory laparotomy yielded diagnosis of sclerosing peritonitis, which resolved after surgery. The short time interval between the processes suggests that they were related to each other, and also to the protein S deficiency.
Subject(s)
Peritonitis/etiology , Phlebitis/etiology , Portal Vein , Protein S Deficiency/complications , Adult , Humans , Male , Peritonitis/pathology , SclerosisABSTRACT
Biopsy proven microhamartomatosis in the liver was demonstrated by sonography and CT in a 72-year-old man with advanced renal failure secondary to autosomal dominant polycystic kidney disease. Sonographic, CT, laparoscopic, and histologic findings are presented.