ABSTRACT
Left renal vein hypertension as a source of persistent gross hematuria has been described in the literature. This type of hematuria is postulated to result from an elevation of left renal vein pressure leading to calyceal-venous communications. We report a case of left renal vein hypertension in which surgical and radiologic findings identify the cause of the hypertension as an aberrant peripheral nerve. To our knowledge, this etiology has never been described. A review of the literature with emphasis on the pathophysiology of renal-vein hypertension is also presented.
Subject(s)
Hematuria/etiology , Hypertension, Renovascular/complications , Renal Veins , Adult , Humans , Hypertension, Renovascular/surgery , MaleABSTRACT
With the advent of more-routine prostate-specific antigen (PSA) blood testing, many more patients are being identified in whom the only clinical sign of their underlying disease is an elevated PSA level. Most evidence to date suggests that such patients do harbor clinically significant cancers and thus should be treated in a fashion similar to patients with T1b, T2a, and T2b disease.
