ABSTRACT
The case of severe skull and brain injury is reported in a patient with known familial paroxysmal hypokalaemic paralysis. Multiple cerebral contusions were seen in the brain trauma. The central nervous function could not be clearly determined since the patient had a hypokalaemic state at the time of admission to the hospital. Only electroencephalographic follow-up studies could reveal the central nervous function at that time.
Subject(s)
Brain Injuries/complications , Hypokalemia/genetics , Paralysis/genetics , Adolescent , Brain Edema/complications , Brain Edema/diagnosis , Brain Injuries/diagnosis , Female , Humans , Hypokalemia/diagnosis , Muscle Hypotonia/diagnosis , Muscle Hypotonia/genetics , Paralysis/diagnosis , Potassium/bloodSubject(s)
Anti-Inflammatory Agents , Dihydrotestosterone/pharmacology , Nitrates/pharmacology , Testosterone/analogs & derivatives , Testosterone/pharmacology , Animals , Cardiovascular System/drug effects , Coronary Vessels/drug effects , Dogs , Female , Male , Pituitary-Adrenal System/drug effects , Rats , Thyroid Gland/drug effects , Vasodilation/drug effectsABSTRACT
1. Dental plaques were detected in all the teeth under investigation, with the exception of impacted teeth. 2. The appearance of the plaques is regular. They occur chiefly in certain zones of the tooth crown and in retention areas. The film thickness ranges from 10 to 60 mum. 3. No alterations of the structure or of the primary texture of the hard tissues were observed on healthy and periodontally diseased teeth. The plaques infiltrate but slightly into the surface. 4. Carious teeth have a different appearance. The plaques infiltrate more or less deeply. They even penetrate into the macroscopically free zones of the enamel as well as of the dentine. The infiltrate is diffuse, especially in regions where the relationship between mineral and organic constituents is distrubed.
