ABSTRACT
A case of localized neonatal herpes simplex virus (HSV) infection involved a prior fetal scalp electrode site. Rupture of the fetal membranes, placement of the fetal scalp electrode and delivery occurred within 30 minutes. The mother had no previous history of genital lesions, and no herpetic lesions were noted at delivery.
Subject(s)
Electrodes , Fetal Monitoring/adverse effects , Herpes Simplex/etiology , Scalp Dermatoses/etiology , Acyclovir/administration & dosage , Acyclovir/therapeutic use , Female , Herpes Simplex/diagnosis , Herpes Simplex/microbiology , Humans , Infant, Newborn , Labor, Induced , Scalp Dermatoses/drug therapy , Scalp Dermatoses/microbiologySubject(s)
Cervix Uteri/microbiology , Streptococcus agalactiae/isolation & purification , Vagina/microbiology , Antigens, Bacterial/analysis , Female , Gram-Positive Bacteria , Humans , Latex Fixation Tests , Predictive Value of Tests , Pregnancy , Sensitivity and Specificity , Staining and Labeling , Streptococcus agalactiae/immunologyABSTRACT
Neonatal septicemia/meningitis from Streptococcus pneumoniae occurred in a 36-hour-old infant. The mother had no overt evidence of infection. This case illustrates the pathogenic potential of this common bacterium in the neonate.
Subject(s)
Meningitis, Pneumococcal/microbiology , Sepsis/microbiology , Streptococcal Infections/microbiology , Adult , Female , Humans , Infant, Newborn , MaleABSTRACT
Using three criteria--(1) herpetic infection and altered organogenesis of inflammatory etiology that antedates dissolution of the fetal membranes, (2) documented herpetic infection present at birth and within 24 hours of rupture of the fetal membranes, and (3) evidence of viral herpetic placentitis the induction of which antedates rupture of the membranes--15 cases of congenital transplacental herpes simplex were identified in the literature. Disease occurring early in gestation produces a cluster of congenital malformations that is clinically indistinguishable from that produced by the cytomegalovirus. Disease manifesting after the shortest recognized incubation period for the induction of the disease in experimental animals does not necessarily preclude transplacental acquisition. Various factors mask the perception of congenital transplacental herpes simplex as a valid disease entity.