ABSTRACT
The so-called safe course of acute myocardial infarction was shown to be associated with reduced left ventricular stroke output, changed phasic pattern of the systole, and peripheral arterial constriction. In infarction-related acute left-ventricular failure, a clear dissociation can be seen between the pumping function of the right and left ventricles. These changes are further aggravated by increased circulating blood volume and pulmonary blood quantity, and lowered arterial and venous flow rate. Paravascular edema adds to generalized vasoconstriction. Hypertension in the pulmonary artery network implies additional effort for right ventricular myocardium and thus prevents blood overflow to lesser circulation veins and left compartments of the heart.
