Use of an equine compression suit to manage a deep axillary wound in a horse.

PURPOSE: To describe the use of an equine compression suit to manage a deep axillary wound in a Thoroughbred filly with bilateral pneumothorax. CASE SUMMARY: A 2-year-old Thoroughbred filly was referred for management of a deep wound to the left axilla. Packing and bandaging of the area were initially attempted, but this became repeatedly dislodged, and bandaging was discontinued. The filly subsequently developed widespread subcutaneous emphysema, and the wound was slow to granulate. Acute onset respiratory distress occurred 11 days after admission, secondary to bilateral worsening pneumothorax, and required placement of a chest drain. A commercially available equine compression suit was then successfully used to hold a primary dressing in place. This resulted in a marked improvement in the subcutaneous emphysema and pneumothorax. Wound granulation subsequently progressed well, and the filly was discharged from the clinic on day 36. UNIQUE INFORMATION PROVIDED: This case report highlights the potential applicability of a compression suit as an alternative to a stent to effectively prevent entry of air and successfully manage axillary wounds in the horse. It was also noted how delayed the progression of a pneumothorax can be after inadequate bandaging of a deep wound in the axillary region. The compression suit provided an alternative means of securing a dressing onto an awkwardly placed wound and may be useful in locations other than the axilla.

Caecal dysfunction following standing surgical procedures.

The aim of this study is to report cases of caecal dysfunction following surgical procedures in the standing horse. The study design is retrospective. Six client-owned horses developed caecal dysfunction following a variety of surgical procedures undertaken in the standing sedated horse. Medical records were reviewed for caecal dysfunctions that had occurred in horses within 2 weeks of standing surgical procedures. Signalment, details of the original standing surgery and medications administered were recorded. Short-term outcome was obtained from clinical records. Long-term outcome was obtained by telephone questionnaire with the owner. Six horses were identified to have developed caecal dysfunction following standing surgery for tooth extraction, laparoscopic ovariectomy, laparoscopic cryptorchidectomy, fracture repair, melanoma removal and castration. Three horses were euthanised with caecal perforation at the time of diagnosis. Three underwent surgical treatment (typhlotomy, decompression and caecal bypass by ileocolostomy). All three horses were alive post-operatively (follow-up at 2, 12 and 24 months). Caecal dysfunction may develop following surgical procedures performed under standing sedation. Careful post-operative monitoring and early identification of caecal dysfunction, and consequent potential need for surgical intervention, are important to optimise outcomes and minimise the risk of fatal caecal perforation occurring.

Serum symmetric dimethylarginine concentration in healthy neonatal Thoroughbred foals.

BACKGROUND: Symmetric dimethylarginine (SDMA) is widely used in other species as a marker of renal dysfunction and is considered a more sensitive indicator of glomerular filtration rate than creatinine. Reference ranges are established in healthy adult horses (≤14 µg/dL) and concentrations are increased in horses with acute kidney injury (median 32 µg/dL; range 15-92). OBJECTIVES: To establish the normal range of SDMA concentrations in neonatal Thoroughbreds. STUDY DESIGN: Cross-sectional. METHODS: Blood samples were collected from Thoroughbred foals <36 h old deemed healthy by physical examination. Exclusion criteria included foals from mares undergoing treatment for placentitis and foals that developed clinical disease or died/euthanised <2 weeks from birth. Biochemistry and serum SDMA concentrations were obtained. RESULTS: Subjects included 120 foals. Median age was 13.5 h (range 1.0-34.0). Median and 95% confidence interval for SDMA concentration was 69.0 µg/dL (63.0, 75.0; range 35.0-376.0). A cut-off value of 168 µg/dL would include 95% of individuals and is therefore suggested. Serum SDMA concentration was correlated with age (R = -.3, P = .003), creatinine concentration (R = .6, P ≤ .001) and urea concentration (R = .3, P = .002). MAIN LIMITATIONS: Limitations include a small sample size, no consideration of subclinical disease and a short follow-up period. CONCLUSIONS: In equine neonates, SDMA concentration is higher than in adult horses, older foals and adults with acute kidney injury. Therefore, currently SDMA cannot be used as a marker of renal dysfunction in this age group. Further work is required to assess whether SDMA concentration is increased in neonates with renal disease and, if so, what cut-off should be used.