ABSTRACT
In the anterior region, the successful replacement of a single tooth with an implant-supported restoration is one of the most difficult treatment options due to numerous functional and biological requisites. Such prostheses should also satisfy the increasing aesthetic demands of patients who expect the definitive restoration to mimic the natural dentition and their supporting gingival tissues. This article highlights the importance of the provisional restoration as the critical element for aesthetic success and gingival integration. Its perfect replication in the definitive restoration requires the use of optimized transference techniques.
Subject(s)
Dental Implants, Single-Tooth , Dental Prosthesis, Implant-Supported , Denture, Partial, Temporary , Esthetics, Dental , Gingiva/anatomy & histology , Adult , Crowns , Dental Abutments , Dental Implantation, Endosseous/methods , Dental Prosthesis Design , Female , Gingiva/physiology , Humans , Incisor , Maxilla , Patient Care PlanningSubject(s)
Crowns , Aluminum , Dental Casting Technique , Dental Porcelain , Denture Design , HumansSubject(s)
Denture Retention , Tooth, Artificial , Adult , Dental Abutments , Dental Cements/pharmacology , Denture Design , Denture, Partial, Temporary , Female , Humans , IncisorABSTRACT
The increase in the maternal plasma A.F.P. level is due to an hypoxia of the foetus. The prospective study of 851 single pregnancies shows that there is a significant rise in the A.F.P. levels during the last days of the pregnancy if the babies are going to exhibit a so-called physiological jaundice at birth: the decrease of the A.F.P. levels in these cases is four times slower than in normal cases. The prospective study of another group of 404 pregnancies gave the same results for the A.F.P. level of the blood of the umbilical cord. Statistical analysis showed that the pathological conditions capable of increasing the A.F.P. levels are related to neo-natal jaundice. The neo-natal jaundice may be due to a factor of foetal hypoxia capable of inducing an over stimulation of the foetal erythropoiesis. The results of this mechanism would be a quantitative disequilibrium between an increased hemolysis and a reduced bilirubine fixation capacity during the neo-natal period.
Subject(s)
Jaundice, Neonatal/blood , alpha-Fetoproteins/analysis , Bilirubin/blood , Erythropoiesis , Female , Fetal Blood/analysis , Fetal Hypoxia/complications , Humans , Infant, Newborn , Pregnancy , Pregnancy Complications, HematologicABSTRACT
When free DHEA, its sulfatide, and sulfate were assayed in maternal plasma as well as in umbilical cord arterial and venous plasma, rather high concentrations were found in either fraction from cord arterial plasma, reflecting the fetal contribution not only of free DHEA and DHEA sulfate, but also of the lipophile steroid sulfatide. Since high DHEA levels were associated with elevated c-AMP concentrations, a certain interrelationship of both parameters is indicated. In the course of delivery, a rapid decrease of free estriol in maternal plasma was observed. Higher concentration of free estriol in umbilical venous plasma pointed at its placental biosynthesis from fetal precursors.
